Answer:
They include somatization disorder, undifferentiated somatoform disorder, hypochondriasis, conversion disorder, pain disorder, body dysmorphic disorder, and somatoform disorder not otherwise specified. 1 These disorders often cause significant emotional distress for patients and are a challenge to family physicians.Nov 1, 2007
Explanation:
the abnormalities the nurse can document during the otoscopic examination are immobility of the tympanic membrane and middle ear effusion.
<h3>What is otoscopy?</h3>
Otoscopy can be defined as a clinical procedure used in the examination of the structures of the ear, particularly the external auditory canal, tympanic membrane, and even the middle ear.
Clinicians carryout otoscopy during routine wellness physical exams and the evaluation of specific ear complaints
During the otoscopic examination, the clinician utilizes an otoscope, also to see through or to visualize the ear anatomy.
The abnormalities that can be documented include;
- Immobility of the tympanic membrane moves in response to pressure.
- The detection of middle ear effusion
Thus, the abnormalities the nurse can document during the otoscopic examination are immobility of the tympanic membrane and middle ear effusion.
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Radioimmunoassay is a test that the nurse expects the physician to order to check thyroid hormones.
The thyroid gland is an important hormone gland that regulates metabolism, growth, and development in the human body. It aids in the regulation of various physiological functions by continuously releasing a consistent amount of thyroid hormones into the bloodstream.
The thyroid gland generates hormones that control the metabolic rate of the body, as well as heart, muscle, and digestive function, brain growth, and bone maintenance.
The most frequent thyroid issues include aberrant thyroid hormone production. Hyperthyroidism is a condition caused by an excess of thyroid hormone. Hypothyroidism is caused by insufficient hormone production.
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Answer:
types 4,8,10
Explanation:
1.
Fibril-forming collagens (I, II, III, V, XI, XXIV, XXVII);
2.
Fibril-associated collagens with interrupted triple helices (FACITs) (IX, XII, XIV, XVI, XIX, XX, XXI, XXII). The FACITs do not form fibrils by themselves but they are associated with the surface of collagen fibrils.
3.
Network-forming collagens (IV, VIII, X) form a pattern in which four molecules assemble via their amino-terminal 7S domain to form tetramers while two molecules assemble via their carboxy-terminal NC1 domain to form NC1 dimers
4.
Membrane collagens (XIII, XVII, XXIII, XXV)
