Answer:
$2,382 / month** $4,764 / month**
Explanation:
Answer:
who is anna , and how would i know did was anna a real person and is she dead
Explanation:
Administer the prescribed enoxaparin (Lovenox).
- The anticoagulant enoxaparin sodium is sold under brand names such as Lovenox. It is used to treat and prevent pulmonary embolism and deep vein thrombosis, especially during pregnancy and after some types of surgery.
- Heart attacks and acute coronary syndromes are also treated. A prescription drug called Lovenox is used to treat and prevent signs and symptoms of chest pain and blood clots (deep vein thrombosis or DVT) (angina). Lovenox can be taken alone or with other medicines.
- Lovenox belongs to a class of drugs known as cardiovascular and blood anticoagulants. Tell your doctor if you have side effects that bother you or that don't go away.
Therefore, enoxaparin must be injected in such cases.
Learn more about blood clots:
brainly.com/question/1501224
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Answer: Biopyschosocial model
Explanation: The biopsychosocial model debates that not any one factor is sufficient; it is the cooperation between people's biology, psychology, as well as social and cultural context that can "interfere" with their health outcomes.
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
