Answer:
TRUE
Explanation:
In the cardiovascular system, the blood flows from <u>high pressure to low pressure.</u>
In the ventricular ejection phase, when the muscles of the ventricles contract, the<u> pressure gradient in ventricles rises</u>. This increases in the <u>pressure in the ventricles exceeds the pressure in the pulmonary artery </u>and aorta, thus opening pulmonary and aortic valves and ejecting the blood from the heart.
Answer: b) Lipogenesis
Explanation:
The postabsorptive state reaction is a reaction which occurs in the state of fasting. In this postabsorptive state the digestive tract remains empty as a result of this the breakdown of the body reserves takes place.
All except the lipogenesis is not the postabsorptive state reaction. This is because of the fact that lipogenesis involves the synthesis of the fatty acids and lipids from typically the carbohydrates and other substances.
Answer:
Very low density lipoprotein (VLDL) is a particle containing triglycerides and cholesterol and protein that is made by the liver. VLDL goes up with diets that contain a lot of fat, sugar, or alcohol. It can also be high in conditions such as diabetes and kidney disease.
Explanation:
Change the environment's temperature or move the patient somewhere warmer. Dry off the patient and the bed sheets.
If the temperature is 32.8°C (91°F). External cooling devices or cold normal saline infusions are utilized to quickly reduce body temperature to 89.6° to 93.2° F (32° to 34° C) when therapeutic hypothermia is used postresuscitation.
What is therapeutic hypothermia ?
- Treatments to lower body temperature include therapeutic hypothermia. This lessens injuries and ongoing issues. It is occasionally applied to patients who experience cardiac arrest. When the heart abruptly stops beating, cardiac arrest occurs.
- The difficulties brought on by hypothermia are intended to be avoided through induced hypothermia. It is mostly utilized in neonatal encephalopathy, head injuries, and survivors of cardiac arrest who are comatose. The prevention of cerebral reperfusion injury is suggested to be a mediator for the mechanism of action.
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