patient with iron deficiency anemia will have low hemoglobin levels as well as microcytic and hypochromic RBCs, according to the results of a complete blood count. Typically, the reticulocyte count is normal or slightly increased.
<h3>what is
iron deficiency anemia?</h3>
- Insufficient iron is the cause of iron deficiency anemia.
- Your body needs iron to produce enough of a component in red blood cells that makes it possible for them to carry oxygen (hemoglobin).
- Thus, iron deficiency anemia may make you feel lethargic and breathless.
<h3>How serious is iron deficiency anemia?</h3>
- Iron-deficiency anemia can have major side effects, including fatigue, headaches, restless legs syndrome, heart issues, problems during pregnancy, and delays in a child's development.
- Additionally, iron deficiency anemia can worsen other chronic illnesses or render their therapies ineffective.
<h3>What three levels of iron deficiency are there?</h3>
Deficiency in Iron Has 3 Stages
- Stage 1: A decrease in the body's overall iron content. A decrease in serum ferritin serves as an indicator of this stage.
- Stage 2 Reduced red blood cell production. This stage develops when there is not enough iron available to sustain the production of red blood cells.
- Stage 3 Iron deficiency anemia.
learn more about Iron deficiency anemia here
<u>brainly.com/question/866200</u>
#SPJ4
"I need to discard unused tablets 6 months after the bottle is opened" means an understanding of this medication.
<h3>
What is PRN?</h3>
This is a latin word which is 'pro re nata' and means as the circumstances arises in this scenario.
Discarding unused tablets 6 months after the bottle is opened is encouraged in this type of medication due to it being taken only when needed.
Read more about Medication here brainly.com/question/26254731
#SPJ12
Answer:
The processing power of the mammalian brain is derived from the tremendous interconnectivity of its neurons. An individual neuron can have several thousand synaptic connections. While these associations yield computational power, it is the modification of these synapses that gives rise to the brain's capacity to learn, remember and even recover function after injury. Inter-connectivity and plasticity come at the price of increased complexity as small groups of synapses are strengthened and weakened independently of one another (Fig. 1). When one considers that new protein synthesis is required for the long-term maintenance of these changes, the delivery of new proteins to the synapses where they are needed poses an interesting problem (Fig. 1). Traditionally, it has been thought that the new proteins are synthesized in the cell body of the neuron and then shipped to where they are needed. Delivering proteins from the cell body to the modified synapses, but not the unmodified ones, is a difficult task. Recent studies suggest a simpler solution: dendrites themselves are capable of synthesizing proteins. Thus, proteins could be produced locally, at or near the synapses where they are needed. This is an elegant way to achieve the synapse specific delivery of newly synthesized proteins.
Explanation:
um I suggest call the ambulance and parents right away...
Answer:
involuntary muscles
Explanation:
Skeletal muscle fibers are cylindrical, multinucleated, striated, and under voluntary control. Smooth muscle cells are spindle shaped, have a single, centrally located nucleus, and lack striations. They are called involuntary muscles.
hope this helps:)
