The mass of the star actually determines the stage of the
star after supernova. If the mass of the star is small, then it would become a
very small and dwarf, cold and dead body in space. The mass of the star has to
be less than that of the sun. If the mass of the star is greater than sun, then
it would become a black hole. The mass of the star has to be more than 1.4
times the mass of the sun to become a black hole.
The goal is to find out how often effective antimicrobial therapy is delayed after the start of persistent or recurrent hypotension in septic shock and how this affects mortality.
Design: A cohort research that was conducted in retrospect between July 1989 and June 2004.
Setting: Ten hospitals (four academic, six community) and fourteen critical care units (four medical, four surgical, and six combined medical/surgical) located in Canada and the United States.
Patients: The 2,731 adult patients with septic shock listed in their medical records.
Measurements and key findings: Survival to hospital discharge served as the primary outcome indicator. A survival percentage of 79.9% was found when an antibiotic efficacious for isolated or suspected infections was administered within the first hour of verified hypotension. Over the following 6 hours, each hour of antibiotic delivery delay was linked to an average 7.6% decline in survival. When compared to obtaining treatment within the first hour after the beginning of persistent or recurrent hypotension, the in-hospital mortality rate was considerably higher by the second hour (odds ratio 1.67; 95% confidence range, 1.12-2.48). The single best predictor of outcome in multivariate analysis (which included Acute Physiology and Chronic Health Evaluation II score and treatment factors) was time to the start of effective antimicrobial therapy. It took 6 hours on average to start effective antimicrobial therapy (25-75th percentile, 2.0-15.0 hrs).
Conclusions: In adult patients with septic shock, effective antibiotic therapy during the first hour of confirmed hypotension was related with enhanced survival to hospital discharge. Only 50% of patients with septic shock got efficient antimicrobial therapy within 6 hours of being diagnosed with proven hypotension, despite a steady rise in fatality rate with increasing delays.
<h3>What is
septic shock?</h3>
Septic shock is a potentially fatal illness that develops after an infection when your blood pressure drops to an unsafely low level. The infection might be brought on by any kind of bacterium.
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Answer:
scientific theory means an idea or proposal of a concept where as an opinion is a thing where u chose something
Explanation:
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Options for the question have not been given. They are as follows:
blood vessel length
blood viscosity
blood vessel diameter
blood colloid osmotic pressure
Answer:
blood colloid osmotic pressure
Explanation:
- Peripheral resistance is the resistance to blood flow by blood vessels.
- It is directly proportional to blood vessel length as more the distance to travel, more will be the resistance.
- It is also directly proportional to blood viscosity as more energy is required to push viscous material.
- It is indirectly proportional to blood vessel diameter. More the diameter of the vessel, easier it is for the blood to pass.
- However, it does not depend on blood colloid osmotic pressure. Colloid osmotic pressure is created by the protein components of blood and they do not have any direct impact on blood flow.
The genetic change causes alterations in only one or a few nucleotide bases in known as mutation.
A mutation is a change in a brief section of a genome's nucleotide sequence. Another frequently replaces one nucleotide in point mutations; other changes involve the insertion or deletion of one or more nucleotides.
Errors in DNA replication or the harmful effects of mutagens, such as chemicals and radiation, which react with DNA and alter the architecture of individual nucleotides, are the leading causes of mutations.
DNA repair enzymes are present in all cells and work to reduce the frequency of mutations. These enzymes reduce genetic information loss, double-strand break formation, and DNA crosslinking by repairing DNA damage.
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