Common signs and symptoms of idiopathic intracranial hypertension (IIH) in the young include headache, vomiting, blurred vision, and diplopia. Clinical manifestations of increased ICP in an infant include a high-pitched cry, poor feeding, setting-sun sign, and distended scalp veins.
This insulin has no peak action and does not cause a hypoglycemic reaction.
<h3>What is
insulin?</h3>
- Insulin is a peptide hormone generated by beta cells of the pancreatic islets and is encoded by the INS gene in humans.
- Its name is derived from the Latin insula, which means "island." It is regarded as the body's primary anabolic hormone.
- It promotes the uptake of glucose from the circulation into liver, fat, and skeletal muscle cells, which controls the metabolism of carbs, lipids, and protein.
- The ingested glucose is transformed in these tissues into either glycogen (through glycogenesis) or fats (triglycerides), or, in the case of the liver, both, via lipogenesis.
<h3>What is the insulin's source?</h3>
- The pancreas, an organ behind the stomach that produces the hormone insulin, is responsible for this.
- The pancreas contains specific regions known as the islets of Langerhans (the term insulin comes from the Latin insula that means island).
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Answer:
Option B.
Explanation:
Cystic fibrosis is caused by a defective gene which causes the lungs and the digestive system to become clogged with sticky mucus resulting the inflammation of the lungs thereby causing difficulty in breathing.
Answer:
The answer to fill in the blank in this question: Some hormones act through cell membrane receptors that stimulate adenylate cyclase activity and production of:____, would be: cAMP.
Explanation:
In the process of hormonal regulation of cells there are two ways in which this can happen: direct activation of DNA inside a cell through steroid hormones, who do not need any kind of mediators to enter the cytoplasm of a cell and activate its genetic material, or indirect activation, which is the common way for non-steroidal hormones, such as epinephrine. In this second scenario a hormone will reach the cell and lock onto a receptor on the plasma membrane. The effect is that a G-protein, which is adjacent to the receptor, activates the second-messenger system, meaning that the activity the hormone came to initiate, will depend on these mediators. the G-protein will then activate adenylate cyclase, which in turn will activate ATP and transform it into cAMP. It is cyclic AMP (cAMP) which will finally relay the original message sent through the hormone, to the genetic material of the cell.
