A maximum of five days due to the hormones most likely leaving your body in that time frame
Answer:
instructs health care providers not to do cardiopulmonary resuscitation (CPR) if a patient's breathing stops or if the patient's heart stops beating.
Explanation:
A recent meta-analysis of seven studies completed that dietary intakes (not vitamin supplements) of vitamin E, C, and beta-carotene existed linked to a decreased risk of Alzheimer disease.
<h3>What is the Causes of Alzheimer?</h3>
In individuals with early-onset Alzheimer's, a genetic mutation may be the cause. Late-onset Alzheimer's arises from a complicated series of brain changes that may happen over decades. The causes probably contain a combination of genetic, environmental, and lifestyle factors.
Symptoms may include:
- Increased memory loss and disarray.
- Inability to discover unique things.
- Problem with language and problems with reading, writing, and performing with numbers.
- Difficulty managing thoughts and thinking logically.
- Compressed attention span.
- Problems managing new situations.
There's currently no treatment for Alzheimer's disease. But there exists medicine available that can temporarily lower the symptoms. Support stands also available to assist someone with the condition, and their family, coping with everyday life.
Hence, A recent meta-analysis of seven studies completed that dietary intakes (not vitamin supplements) of vitamin E, C, and beta-carotene existed linked to a decreased risk of Alzheimer disease.
To learn more about Alzheimer refer to:
brainly.com/question/27414232
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Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
