I have searched everywhere, but I have not found the proposals of the question, but I will explain to you what is the endoplasmic reticulum so that you can answer it.
The endoplasmic reticulum is a eukaryotic organelle located in the cytoplasm.
The endoplasmic reticulum is a network of membrane tubules (often interconnected) scattered throughout the cytoplasm of eukaryotic cells. Its membrane, which alone represents more than half of the cellular membrane system, is in contact with the nuclear envelope.
The endoplasmic reticulum can be:
Granular (or rough) (RER) that is to say associated with ribosomes.
Smooth (SER).
The granular endoplasmic reticulum is the place of synthesis (in the associated ribosomes) of the proteins secreted outside the cell and of the proteins and lipids constituting the membranes of the cellular organelles. Golgi, lysosomes, mitochondria, nucleus, ribosomes, vesicles ...). It participates in the correct folding of the proteins that have just been synthesized.
The smooth endoplasmic reticulum participates in cellular metabolism, synthesizing lipids and storing calcium.
The cell goes in a "dormant phase". A good example is the neuron. Neurons are very metabolically active but they don't divide so they go into a phase that doesn't include growth or replication of genetic material.
Answer:
Taxol induces the assembly of microtubules, while Nocodazole suppresses tubulin assembly
Explanation:
Taxol and nocodazole are antimitotic drugs, i.e., drugs that inhibit the progression through the cell cycle, which is useful for killing cancer cells. Microtubules are proteins that act as the 'skeleton of the cell', which need to be reorganized when cells replicate. These structures (microtubules) are composed of α and β-tubulin heterodimers which assemble into protofilaments of microtubules. Taxol is an antimitotic drug that induces the formation of microtubules, thereby inhibiting microtubules' reorganization during mitosis. In consequence, cells treated with Taxol enter into apoptosis (i.e., programmed cell death). Moreover, Nocodazole is known to induce microtubule disassembly by interfering with the polymerization of tubulin monomers. In consequence, Nocodazole is useful to depolymerize the microtubule cytoskeleton.
D.uracil is the nitrogen base bind with adenine
