Answer: adenylyl cyclase inositol triphosphate mainly known as IP3 causes the release of Calcium ions directly from the inracellular stores and causea contraction.
Explanation:
IP3, inositol phosphate is a second messenger a signaling molecule. It is made by hydrolysis of phosphatidylinositol 4,5-bisphosphate (PIP2), a phospholipid that is located in the plasma membrane, by an enzyme phospholipase C.
IP3 binds to the calcium channels and opens Ca2+ channels that are embedded in the ER membrane, releasing Ca2+ into the cytosol. Calcium ions released may cause contraction and regulate the Ca2+ channels in the membranes.
The answer would be D. variation
Answer:
No, CREs usually don't account for differences in gene expression levels.
Explanation:
<em>Cis</em>-regulatory elements (CREs) are non-coding DNA regions that regulate adjacent gene <em>transcription</em>. They usually do it by binding to transcription factors. They are in charge of influencing the rate of which transcription occurs, and less with how much a gene is expressed. On the other hand, trans-regulatory elements are DNA sequences that encode for upstream regulators that can modify or regulate <em>gene expression levels</em>.
Therefore, cis elements are not really in charge of expression levels, mostly transcription rates of single genes, rather. Trans elements are more involved in expression levels.
Answer:
I am from India
Explanation:
you are free you are from here
Answer:
Abstract
Respiratory homeostasis is concerned with the regulation of a blood gas composition that is compatible with maintaining cellular homeostasis. Provided that the lung-capillary exchange barrier does not prevent the exchange of gases, then blood leaving the lung will have oxygen and carbon dioxide partial pressures that are similar to the average values found in the alveoli. Alveolar ventilation establishes these values. If blood gas composition, especially of carbon dioxide, moves outside the homeostatic range, the change is detected by chemoreceptors and respiratory responses are promoted which change alveolar ventilation, alter alveolar gas composition and so reverse the change. Ventilation therapies provide the means of artificially restoring alveolar gas composition. In general terms, they do this by raising the partial pressure of oxygen within the alveoli either by using oxygen-enriched gas mixtures, or by improving the ventilation of alveoli using positive pressure.
Explanation: