Answer:
<u> The following four traits are -: </u>
- <u>Pedigree 1 -</u> A recessive trait (autosomal recessive) is expressed by pedigree 1.
- <u>Pedigree 2- Recessive inheritance is defined by Pedigree 2. </u>
- <u>Pedigree 3</u> - The inheritance of the dominant trait (autosomal dominant) is illustrated by Pedigree 3.
- <u>Pedigree 4-</u> An X-like dominant trait is expressed by Pedigree 4.
Explanation:
<u>Explaination of each pedigree chart</u>-
- Pedigree 1 demonstrates the <u>recessive trait </u>since their children have been affected by two unaffected individuals. If the characteristics were X-linked, in order to have an affected daughter, I-1 would have to be affected. In this, both parents are autosomal recessive trait carriers, so the child will be affected by a 1/4 (aa)
- <u> Recessive inheritance</u> is defined by <u>Pedigree 2</u>. This is<u> X-related inheritance as autosomal recessive</u> inheritance has already been accounted for in part 1. This inference is confirmed by evidence showing that the father (I-1) is unaffected and that only the sons exhibit the characteristic in generation II, suggesting that the mother must be the carrier. The individual I-2 is a carrier for this X-linked trait. A typical Xa chromosome is attached to the unaffected father (I-1), so the chance of carrier II-5 is 1/2. Probability of an affected son = 1/2 (probability II-5 is a carrier) x 1/2 (probability II -5 contributes () x 1/2 (probability of Y from father II-6) = 1/8. An affected daughter's likelihood is 0 because a typical must be contributed by II-6.
- The inheritance of the<u> dominant trait</u> is demonstrated by <u>Pedigree 3 </u>because affected children still have affected parents (remember that all four diseases are rare). The trait must be <u>autosomal dominant</u> because it is passed down to the son by the affected father. There is a 1/2 risk that the heterozygous mother (II-5) would pass on mutant alleles to a child of either sex for an autosomal dominant feature.
- <u>Pedigree 4</u> is an <u>X-linked dominant function</u> characterized by the transmission to all of his daughters from the affected father but none of his son. On the mutant X chromosome, the father (I-1) passes on to all his daughters and none of his sons. As seen by his normal phenotype, II-6 therefore does not bear the mutation. An affected child's likelihood is 0.
In the question the pedigree chart was missing ,hence it is given below.
No, although there are several theories with increasing evidence, there is still no way to know for sure how it occurred. The great mystery of the beginning of universe is way too vast and vague. Theories can only be proven by presenting a solid proof, but great creations such as universe itself doesn't have to be solved. Various possibilities must have happened or there must be a one conclusion leading to its existence; but moreover, it is not our problem to solve. The confirmation of beginning of its existence will only feed the curiosity of humans.
Sandstone rocks<span> are most likely the result of compacting and cementing particles together.</span>
CHO is the short term for carbohydrate in medical lingo.
So it would be Carbon, Hydrogen and Oxygen
A. Carbon is the answer