Answer:
they are all things related to decomposed corpse remains or like remains in a animal.
Explanation:
Temperature affects spermatogenesis, which functions best at body temperatures just a little lower than those.
<h3>Abstract:</h3>
To keep testicular temperatures below those of the body core, adequate thermoregulation is essential. The process of mammalian spermatogenesis and the resulting spermatozoa are negatively impacted by elevated testicular temperature. Therefore, sperm quality can be affected and the likelihood of infertility is increased by thermoregulatory dysfunction resulting in heat stress. This article reviews a variety of internal and external factors that may lead to testicular heat stress. We go into more detail on how heat stress affects the spermatogenesis process, the resulting epididymal spermatozoa, germ cells, and the alterations that result in the testis.
We also go over the chemical reactions of germ cells to heat exposure and potential processes, such as apoptosis, DNA damage, and autophagy, that could lead to heat-induced germ cell damage. Further explanation is provided for the intrinsic and extrinsic processes involved in the complex mechanism of germ cell death. These intricate apoptotic pathways ultimately result in the demise of germ cells.
Learn more about spermatogenesis here:
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Answer:
Natural selection disrupts the genetic equilibrium by favoring the genes for the traits that impart survival advantage to individuals.
Explanation:
Natural selection refers to the unequal survival rate of individuals of a population due to the presence of some beneficial adaptive genetic trait. These individuals are able to survive more and leave more progeny. Over generations, the population has increased number of these individuals leading to evolution of a population with different allele frequencies.
For example, the frequency of gene for antibiotic resistance in the bacterial population was increased once humans started using antibiotics to control the bacterial population. This led to the evolution of the bacterial population with antibiotic resistance.
Is this school work ? can you tell me so i can tell you professical or common
Question is incomplete i have added full question in ask for detail section as well as in picture format with answer. See figure for clear explanation.
Answer:
(Twisted leaves)
The mutant allele is dominant to its corresponding wild-type allele
(Forked leaves)
The mutant allele is dominant to its corresponding wild-type allele
(pale leaves)
The mutant allele is neither dominant nor completely recessive to its corresponding wild-type allele
Explanation:
I have attached picture which explains phenomenon it self.