Answer: <u>Both parents are heterozygous carriers</u>.
Explanation: Since sickle cell disease is a recessive trait, it means the affected person will have "homozygous recessive" state of genotype only. Here parents are normal, which means they are carriers. So their genotype would be Ss, heterozygous.
Heterozygous parents only will have 25% chances of having their children affected.
Option 1 is incorrect, because if one parent has two recessive alleles, it means he/she is affected and this contradicts the situation mentioned in question.
Option 3 and 4 are incorrect because it is mentioned in question that both parents are normal.
Answer:
a b and f
Explanation:
yep it has all these resources because it just does
Answer: Lateral prefrontal cortex
Explanation: Lateral prefrontal cortex (LPFC) is a region of frontal cortex in the brain, where varying goal-directed behavior is expressed.
Therefore, the region have strong connections with motor nerve cells and are activated by procedures or tasks that requires high-level cognition.
However, presentation of juice in the mouth following a spike in neural activity reinforces the lateral prefrontal cortex (LPFC) to encodes information about the response in the Japanese monkeys.
Muscle cramps are caused when the muscle takes longer than normal to relax after a contraction. During contraction, the myosin heads attach on their binding sites on the actin filament and pulls on the filament hence shortening the sarcomeres. For the myosin head to release its binding site on the actin filament, ATP attaches to an active site on the myosin and is hydrolyzed to ADP and Pi. This causes relaxation after a contraction cycle. Cramps, therefore, may be due to depletion of ATP molecule in the muscles cells.
Another reason for cramps may be a high amount of calcium in the muscles that bind to troponin on the actin filaments. This exposes the myosin binding sites longer hence promotes the formation of cross-bridge even when the contraction of the muscle is not intended anymore.
