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Travka [436]
3 years ago
12

Which of the following correctly describes Cytokinesis? A. The Cytoplasm splits, forming two daughter cells B. Mitotic spindle a

ttaches to the chromosome's centromeres and lines them up in the middle of the cell. C. Centrioles move to opposite sides of the cell, nuclear envelop dissolves, chromosomes form D. Chromosomes split and move to opposite sides of the cell, the cell elongates
Biology
1 answer:
Serggg [28]3 years ago
4 0

The correct answer is A. The cytoplasm splits, forming two daughter cells.

Cytokinesis occurs after the process of mitosis and is the splitting of the one large cell into two separate daughter cells. Cytokinesis is the last step of the cell cycle, after which the cell reenters interphase and begins preparing for its next division. Cytokinesis occurs differently in different types of cells: typically by cleavage in animal cells but by a cell plate in plant cells.

Hope this helps!

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3 0
3 years ago
One gamete is formed during:
Anarel [89]

Answer: Oogenesis

Explanation:

Oogenesis is a process in female reproductive system in which a matured ovum is formed from egg cell.

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There are three stages of Oogenesis, the multiplication phase,the growth phase and maturation phase.

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5 0
3 years ago
Read 2 more answers
Which of the organelles below would you expect to find in all living cells?\
Greeley [361]
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7 0
3 years ago
Name four functions of proteins
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5 0
3 years ago
dynamic modeling of alpha-synuclein aggregation for the sporadic and genetic forms of parkinson's disease
Triss [41]

Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.

We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.

Learn more about Parkinson's disease brainly.com/question/28169444

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3 0
2 years ago
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