I believe it would be the bottleneck effect. hope that helps =)
When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
Answer:
True
Explanation:
Assumption of risk is a defense in the rule of offences, which bars or decreases a plaintiff's right to retrieval in contradiction of a inattentive tort feasor if the offender can prove that the plaintiff willingly and meaningfully expected the hazards at subject inherent to the unsafe action in which the plaintiff was contributing. The most shared specimen is a waiver of obligation employed earlier contributing in a hazardous action. Frequently at matter in belongings where the respondent gifts an rapid statement of the danger defense is whether the plaintiff decided to assume the risk of the specific harm that happened.
Answer: No
Explanation: It is beneficial if the animals that are disadvantageous doesn't reproduce so it doesn't discredit natural selection. The traits in which variation is occured are more suitable for organism to live and reproduce in the environment are preserved and are passed from generation to generation.
However, natural selection effects limited number of progeny.
