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3 years ago

Ture or false: smoking causes early wrinkles and premature age

Biology

2 answers:

Tanya [424]3 years ago

5 0

True would be ur answer

cluponka [151]3 years ago

5 0

Truth, smoking is one of the strongest predictor of face wrinkles in men and women, many more then 4000 chemicals in tobacco smoke also cause damage to collagen and elastin, which which are fibers that give your skin the strength and elasticity.

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A 4-year-old child who has never been separated from parents or siblings is admitted to the hospital. what is most important for

ahrayia [7]

<span>Since the child has never been away from his parents and siblings and also he is only 4 years old it is better to have one of them stay with the child. The presence of a family member will reinforce trust and provide support to the kid.</span>

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3 years ago

Where would a primary producer be located in a food chain

Novosadov [1.4K]

At the base of the food chain

7 0

2 years ago

Read 2 more answers

Provide an adaptive and a nonadaptive hypothesis for the evolutionary loss of useless organs, such as eyes in many cave-dwelling

Zepler [3.9K]

In order to be able to Provide an adaptive and a non-adaptive hypothesis, the following is required

<h3>What is adaptation?</h3>

It refers to a trait or an integrated set of traits that increases the fitness of an organism. It is the process of improving the fit of phenotype to environment through natural selection.

<h3>What is non adaptation?</h3>

it refers to those traits that do not result in the better adaptation of the organism to its environment, and does not increase the genetic fitness of the organism.

Let us take the case of vain organs. There may be a metabolic value to constructing a vain organ, and subsequently the humans who have misplaced that organ might also have a selective advantage. Also, if the organ is useless, the mutations that disrupt the improvement of that organ would be evolutionarily neutral, and may want to unfold to fixation via genetic drift.

So an adaptive hypothesis as to why an organism may lose useless organs is energy trade offs. The organism may additionally be losing energy in growing and retaining a vain organ, whilst this strength can be higher used someplace else. So an organism that lives in a darkish cave might also sooner or later lose its eyes considering eyes are of no use inside a darkish cave.

A non adaptive hypothesis for the evolutionary loss of organs can be random mutation. If a random mutation occurs that causes an organism to lose its eyes, in a dark cave where it does not use its eyes anyway, it could still survive, reproduce and pass that mutation down.

For more information on adaptive hypothesis, visit

brainly.com/question/975242

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3 years ago

Name three cell appendages

mel-nik [20]

The answer is a slim layer, Flagella, and a Fimbriae

Hope this helps!!

5 0

3 years ago

Identify the true statements regarding liver glycogen phosphorylase a. Protein phosphatase 1 is abbreviated PP1. The binding of

katrin2010 [14]

Answer:

Liver phosphorylase a concentration decreases when glucose enters the blood.

The binding of glucose to liver phosphorylase a shifts the equilibrium from the active form

As the concentration of phosphorylase a decreases, the activity of glycogen synthase increases. to the inactive form

Explanation:

Protein phosphatase 1 (PP1) is a phosphatase enzyme known to remove phosphate groups from serine/threonine amino acid residues. PP1 plays diverse biological roles including, among others, cell progression, control of glucose metabolism, muscle contraction, etc. In glucose metabolism, PP1 regulates diverse glycogen metabolizing enzymes (e.g., glycogen synthase, glycogen phosphorylase, etc). In the liver, glycogen phosphorylase catalyzes the rate-limiting step in glycogenolysis by releasing glucose-1-phosphate. Glycogen phosphorylase <em>a</em> is converted (and inactivated) into the <em>b</em> form by PP1, which catalyzes the hydrolysis of the phosphate bond between serine and the phosphoryl group. In the liver, glucose binds in order to inhibit glycogen phosphorylase <em>a</em>, thereby inducing the dissociation and activation of PP1 from glycogen phosphorylase <em>a</em>.

5 0

3 years ago