Answer:cell wall yes plant cell no animal
mitochondria yes both
chloroplasts yes plant cell no animal cell
nucleus yes both
Vacuoles yes both
Lysosomes yes both
Explanation:
Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
This is false for a number of reasons. While LH (which stands for Leutinizing Hormone) is released by the anterior pituitary gland and can be present in both females and males, it play a more predominant role in females. Working closely with FSH (Follicle Stimulating Hormone) a surge in LH begins the process of ovulation and the development of the Corpeus Leuteum.
In Males it is used both for the production of sperm but to stimulate the production of testosterone.
Their mass because it can let you know their weight and size
