The correct answer is: the short half-life of edrophonium makes it impractical for long-term.
Edrophonium is used for the diagnostic of myasthenia gravis. In patient with myasthenia gravis, the body produces autoantibodies which inhibit nicotinic acetylcholine receptors in the neuromuscular junction. Edrophonium, on the other hand, is acetylcholinesterase inhibitor which blocks the effect of acetylcholinesterase enzymes (AcH stays in synaptic cleft).
Substitution for the edrophonium is pyridostigmine which is also acetylcholinesterase inhibitor but with long-term maintenance.
Wouldn't the population then become a mix of both turning into purple.
Answer:
The promoter region/sequence
Explanation:
Promoter sequences/region is where the transcription factors and RNA polymerase must attach before the gene can be transcribed. SO if we mutate this sequence, the transcription factors and RNA polymerase won't recognize it thus would not attach and transcribe the targeted gene.
A ) if it s dna G with C and A with T so if it s C 32 G its 32 you have only 100% so it should be 100-32-32 =36 A=T so 36=A+T 64=2T T =18
Answer:
The models are missing in the question. The models along with the question is provided in the attachment below.
The answer is : Model A represents the Plasma membrane.
Explanation:
According to the question, model A represents the plasma membrane. The reasons are :
(a). In the model A, the bilayers consists of two leaflets of the amphipathic lipid molecules. Here the polar head groups is in contact with the intra cellular or the extra cellular non aqueous phase, where the non polar tails are facing each other constituting the hydrophobic interior of the membrane.
(b). Lipid are not rigid as well as static structure. In the lipid bilayer, the lipid molecule are able to rotate around their axis freely and diffuse laterally with each leaflet.
(c). The plasma membrane acts as the selective plasma membrane.