Answer:
White: A---
Black: aaB-
Brown: aabb
Explanation:
As per given information, the genotype of parent white sheep is AaBb (heterozygous white).
The gene "A" is dominant over gene "B". Hence, all the genotypes with one or two copies of "A" would exhibit white phenotype.
All the genotypes that have "aa" and one or two "B" alleles would exhibit black phenotype.
All the genotypes with "aabb" allele combination would exhibit brown phenotype.
Answer:
they are all heterotrophic
The monarch butterfly population has declined due to a scarcity of :
<h3>The Monarch Butterfly </h3>
Monarch Butterflies are one of the most beautifully colored butterflies with yellow and black lines, they migrate seasonally during the winter, Caterpillars of monarch butterfly feed on the leaves of milkweed which is a major source of food for Monarch butterflies.
The scarcity of milkweed will result in the decrease of the population of Monarch butterflies as the caterpillar will have nothing to feed on .
Hence we can conclude that the monarch butterfly population has declined due to a scarcity of milkweed.
Learn more about Monarch butterfly : brainly.com/question/289820
Solution:
Fetal programming occurs during embryonic and fetal development, a critical period in which tissues and organs are created. Insufficient nutrition during this time results in permanent alterations to certain structural and physiological metabolic functions of the fetus. British epidemiologist Barker first established the hypothesis, known as the "Barker hypothesis,” which states such programmed changes during this critical period predispose the fetus to certain postnatal diseases. The critical period coincides with the timing of rapid cell differentiation. Essentially, it refers to the process of sustaining or affecting a stimulus or impairment that occurs at a crucial point in its development.
Rickets has long demonstrated that under nutrition in the critical early stages of life brings about a continuing change in structure. A recent new doctrine suggests that fetal programming can affect diseases in adulthood. That is, the body's “memory” of under nutrition during the early stages of development translates into a pathology that determines future diseases. This idea is based on animal studies that demonstrate how under nutrition in utero can alter blood pressure, cholesterol metabolism, insulin response to glucose, and other metabolic, endocrine, and immune functions important to human diseases. This paper reviews evidence of the correlation between fetal under nutrition and diseases found in previous studies and considers the mechanism of fetal programming and the role of the placenta.