1. rise of angiosperms
2. rise of chemoautotrophs and photoautrophs
3. rise of multicellularity
4. rise of bryophytes
5. rise of gymnosperms
6. rise of eukaryotes
7. rise of cyanobacteria
Answer:
The options
A)Damage to cellular mitochondria
B)Increased ATP levels
C)Activation of the p53 protein
D)Apoptosis
The CORRECT ANSWER IS D
D)Apoptosis
Explanation:
The extrinsic pathway of apoptosis includes extracellular signaling proteins which adhere to cell surface molecules known aa death receptors which in turn activates apoptosis.
The aftermath activates endonucleases prompting division of DNA and ultimately cell death.
Apart from the TNF and Fas ligand, primary signaling molecules also promotes the extrinsic pathway, examples of such are the TNF-related apoptosis-inducing ligand (TRAIL); the cytokineinterleukin-1 (IL-1); and lipopolysaccharide (LPS), the endotoxin located in the outer cell membrane of gram-negative bacteria while the activation of the p53 protein, and decreased ATP levels in the intrinsic pathway results in DNA damage.
In hemoglobin, the transition from t state to r state (low to high affinity) is triggered by Bisphosphoglycerate (BPG)
- Bisphosphoglycerate (BPG), also known as 2,3-Disphosphoglycerate (2,3-DPG), aids in the transition of hemoglobin from a high-oxygen-affinity to a low-oxygen-affinity state.
- 2,3-BPG binds to hemoglobin, causing oxygen to be unloaded. Furthermore, 2,3-BPG reduces hemoglobin's affinity for oxygen. As hemoglobin is unloaded in our tissues, 2,3-BPG binds to it, promoting oxygen unloading.
- When we increase the concentration of 2,3-BPG in our blood, the oxygen binding curve shifts to the right. This means hemoglobin will have a lower affinity for oxygen and will be able to deliver more oxygen to our body's tissues and cells.
Learn more about Bisphosphoglycerate (BPG) from here:brainly.com/question/8885734
#SPJ4
Answer:
1.The answer is that because of the process of meiosis and fertilizations
Explanation:
