E<span>ach atomic number identifies a specific element, but not the "Ion/Charged particle"
Hope this helps!</span>
Why are people just giving away the answer?
Question: Is the pre-synaptic or the post-synaptic neuron damaged? Explain your rationale in detail (in other words, detail how you came to your conclusion). You're going to have to really think on this one. It's not something you can look up in the text. You will need to use what you have learned about the autonomic nervous system and some critical thinking to arrive at the answer.
Horner's Syndrome riddle: Ach is the neurotransmitter at all ANS preganglionic fibers, including SNS. The pt has decreased sweating and pupil constriction, indicating blockage of SNS transmission. When the MD gave ACh it induced sweating, but not pupil dilation. This is because the sweat glands are an exception in normal SNS transmission, as the postganglionic fibers utilize ACh as their neurotransmitters. Other SNS pathways utilize NE as their neurotransmitter. So ACh acted directly on the sweat gland to produce sweating, but could not stimulate the typical postsynaptic neurons to release NE. When the MD gave NE he saw pupil dilation, but not sweating, indicating the NE worked directly at the iris to produce pupil dilation. This indicaates the problem was with the postsganglionic neuron. If the preganglionic neuron were the problem injecting ACh would have solved both problems.
Answer:
They regulate the cell cycle by phosphorylating other proteins
Explanation:
they are serine-threonine kinases, they play an important role in the phosphorylation of other proteins. Cyclin-CDK complexes phosphorylate substrates appropriate for a particular cell cycle phase and without that, the cycle cannot continue.
