Explanation:
The eye receives oxygen through the aqueous. Its function is to nourish the cornea, iris, and lens by carrying nutrients, it removes waste products excreted from the lens, and maintain intraocular pressure and thus maintains the shape of the eye. This gives the eye its shape. It must be clear to function properly.
Herpes simplex, type 2 is typically the strain associated with genital herpes. A symptomatic patient with herpes may describe vulvar lesions causing pain with sitting. When lesions are visible, herpes is characterized by multiple, painful vesicles and/or ulcerated lesions. Herpes lesions are typically much smaller than the described single mass.
<span>Lichen sclerosis is a chronic, progressive dermatological problem; it often involves the vulvar and perineal epithelium. It is characterized by pruritus, dyspareunia, and fissuring of the skin. Lichen sclerosis is more common in post-menopausal women, and physical exam reveals thin, white-appearing tissue. Masses are not associated with lichen sclerosis.</span>
Answer:
The difference is that the ones with dots have more cells in them and the other one must've been left there for awhile
Explanation:
Because i looked up what those dots meant
Answer:
I wish I could help but do not understand one bit like I am lost right now my brain is dead I can't get one thing you write
<h2>CRISPR/Cas9</h2>
Explanation:
CRISPR can be used to reintroduce dystrophin back into the KO mouse
- CRISPR stands for Clustered Regularly Interspaced Short Palindromic Repeats and is used to for gene editing
- CRISPR/Cas-mediated genome editing has been shown to permanently correct DMD mutations and restore dystrophin function in mouse models
- Germline editing by injecting zygotes with CRISPR/Cas9 editing component was first done in mdx mice by correcting the mutated exon 23
- Postnatal editing of mdx mice was then achieved using recombinant adeno-associated virus to deliver CRISPR/Cas9 genome editing components and correct the dystrophin gene by skipping or deleting the mutated exon 23 in vivo
- Germline and postnatal CRISPR/Cas9 editing approaches both successfully restored dystrophin function in the mice and same technique can be used for KO mouse model
