Answer:
All of the options are true for a MRSA infection.
Explanation:
<em>Staphylococcus aureus</em> is one of the most frequent pathogens causing hospital and community infections. <em>S. aureus</em> can become very easy methicillin resistant (called MRSA isolates) and others beta-lactam antibiotics (are the ones widely used to treat infections) and usually can be resistant to other class of antibiotics, become a very strong bacteria making treatment options very limited. MRSA isolates can rapidly transfer the methicillin resistance to other species of S<em>taphylococcus</em> and some other bacteria. Also <em>S. aureus</em> can acquire other antibiotic resistant genes making a deadly bacterium for its strong resistance. It is in search how the bacterium acquire this antibiotics resistance ( and other virulence factors genes) and the mechanism involve to develop new drugs to treat MRSA infections with the hope that can´t develop resistance to this new drugs.
An ecosystem is a geographic area where plants, animals, and the other organisms work for the existence of bubble life.
<h3>
Variation of ecological habitats within a species is characteristic of which type of diversity?</h3>
The variation of ecological habitats within a specie is character of the type of diversity is known as Ecosystem Diversity.
Ecosystem is defined as the different kinds of habitats, ecological processes and communities. A biological community is defined by the specie which occupy a specific area and the interactions between the species.
So we can conclude that: An ecosystem is a geographic area where plants, animals, and the other organisms work for the existence of bubble life.
Learn more about Ecological Habitat here: brainly.com/question/7413811
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Answer:
No, telomerase is not an oncogene. It prevents the senescence that would occur due to shortened telomeres, but the cell proliferation might still be mitogen-dependent.
Explanation
telomerase is not responsible for causing deregulation while oncogenes cause deregulation .
Telomeres length shorten after the cell division which stops them to divide again and cell die.
Telomerase prevents this decline in some kinds of cells, including stem cells, by lengthening telomeres, and the hope was that activating the enzyme could prevent senescence.
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