The shape of chromatin, which can be either open (euchromatin) or compact (heterochromatin), is dynamically regulated during the phases of the cell cycle is the two types of conformations.
- The main distinction between conformation and configuration is that whereas the configurations of the same molecule do not easily interconvert, their conformations do.
- With a predefined location in the nucleus and a certain form, such as metacentric, submetacentric, acrocentric, or telocentric, chromosomes are primarily heterochromatic in this stage.
- All DNA-mediated processes, including gene regulation, can be significantly impacted by the degree of nucleosomal packaging.
- While heterochromatin (tight or closed chromatin) is more compact and resistant to factors that need to access the DNA template, euchromatin (loose or open chromatin) structure is permissible for transcription.
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The specific volume will be different for various kinds of cells. The safe answer would be that the new cell will pretty much have the same volume as the one that it divided from. This is true for most eukaryotic cells unless other factors like epigenetics or mutations come into place.
One example of moments a cell would increase in volume is during hypertrophy. This simply means that the cell is increasing in size (compared to: hyperplasia -- which is an increase in number of the cells). Hypertrophy is definitely an increase in volume of the cell but this doesn't necessarily translate to cell division (i.e. just because the cell is big now, doesn't mean it will still be big when it divides).
Another moment of increasing volume of the cell and now also related to cell division would be during the two stages in the cell cycle (i.e., G1 and G2 phases). This is the growth phase of the cell preparing to divide. However when mitosis or division happens, the cells will normally end with the same volume as when it started.
This are safe generalizations referring to the human cells. It would help if a more specific kind of cell was given.
Answer:
A titin mutation that occurs in muscular dystrophy with myositis (mdm) mice results in a predicted 83 amino acid deletion in the N2A and PEVK regions of the titin protein. Muscles from mdm mice are actively more compliant possibly owing to the deletion in titin's I-band region. This suggests that modulation of titin stiffness in active sarcomeres by the proposed titin–thin filament interaction may be affected by the mdm mutation. The answer is YES I believe.
Explanation:
I believe the answer is yes from my deep reaserch. You may want to research in your texts book/lesson or courses and review what your teacher/professer has given you.
Only the energy used for growth (33 J) is available to the next trophic level, because it is used to produce more biomass which can be consumed by the next trophic level.
