Answer:
<h2>As transcription and translation occurs in different location in eukaryotes, while in prokaryotes, both the processes occurs at same location, post-transcriptional processing in eukaryotes. </h2>
Explanation:
The transcription and translation occurs in different location in eukaryotes, while in prokaryotes, both the processes occurs at same location and can be co-translation.
As, in eukaryotes, the transcription occurs in the nucleus and then this transcript have to move in the cytoplasm to translation, so before reaching in the cytoplasm, various modifications occur in this transcript known as post transcriptional modification or processing ( include 5' capping, 3' poly-adenylation and RNA splicing).
As, in prokaryotes, both transcription and translation occur at same place so there is no time for modification for transcript because translation starts as the transcription is still going on.
We sprinkle powder on carrom board to make the surface of the board smooth. This reduces the friction between the surface of the carrom board, the striker and the coins. As a result, the coins and the striker can move easily on the carrom board.
* More than 40 proteins and glycoproteins involved in the complement system are synthesized by the liver, macrophages, epithelial cells, they are present in the blood in plasmatic form, membrane, some have an enzymatic activity, regulator or membrane receptorThese are elements of the humoral innate immune response, they fight infections, purify immune complexes and apoptotic bodies.
<span>There are indeed three ways to activate the complement:</span>
Classical pathway: Activated by Immunoglobulins in immune complexes, aggregated Immunoglobulins, DNA, CRP, apoptotic bodies .......it involves nine fractions, starting with C1, then C4, C2, C3, to form a classical C5 convertase, then, activation of C5, C6, C7, C8, C9.
Alternative pathway: activated by polysaccharides (bacterial endotoxin), vascular wall poor in sialic acid, aggregated IgE ...C3b like is the first component in the alternate channel cascade, it will create an amplification loop, and form an alternative C5 convertase.
Lecithin pathway: Activated by mannose, fucose (carbohydrate of microorganisms)The first component is the complex MBL / MASP1 / MASP2: "mannose-binding protein": works according to the same principle as the complex C1 of the classical way (MASP2 cleaves the C4 and the rest of the cascade is equivalent to that of the classical way).
the three ways have the same outcome: A C5 convertase (formed by one of the pathways) cleaves C5 into C5a and C5b: C5b is deposited far from other fractions on the antigenic surface. The fixation of C5b in the cell is followed by that of C6, C7, C8, and C9 (9 molecules of C9): formation of the membrane attack complex (MAC) ==> Death of the cell by osmotic shock
Answer:
plate boundaries
Explanation:
i just think i remember it from 6th grade Imao, its been a couple years though
Answer:
Copper (II) fluoride
Explanation:
Copper (II) fluoride b/c fluoride is charge -1, so if its F2 that must mean copper was +2.
Transition metal-nonmetal nomenclature:
Metal name + (charge in roman numeral) + non-metal_ide
