The effects of a right hemisphere stroke may include: Left-sided weakness or paralysis and sensory impairment. Denial of paralysis or impairment and reduced insight into the problems created by the stroke (this is called "left neglect") Visual problems.
<h3>What is impacted by a stroke in the right hemisphere?</h3>
A right hemisphere stroke may result in sensory impairment and left-sided weakness or paralysis. Denial about paralysis or impairment as well as diminished awareness of the problems caused by the stroke are referred to as "left neglect." Visual issues., such as an inability to see each eye's left visual field.
<h3>Does a stroke on the right side impact speech?</h3>
Speech and communication issues are typically seen in right-brain injured stroke survivors. Because of their weak or uncontrolled left side facial and mouth muscles, many of these people have trouble pronouncing spoken sounds correctly. Dysarthria is the term for this.
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Answer:
The answer to the question: Where does the stimulus occur in order to initiate an AP, would be, B: Dendrites.
Explanation:
When an impulse is to be generated and passed on as an action potential towards a corresponding neuronal cell, and a final affected organ, the neurons need first to be stimulated so that an action potential begins. This stimulus comes as a neurotransmitter released by other neurons near the one that will be stimulated. This neurotransmitter will bind to the receptors on the dendrites of the neuron to be stimulated and immediately this will cause the ion channels, gated and non-gated, to open and close so that an action potential can be initiated. The cell body then initiates the first action potential, and will in turn stimulate the axon to also start their own action potential, which will, like a domino effect, move down to the axon terminals. This process will be followed all along a neuronal circuit.
Answer:
advocacy, public support, or encouragement
Explanation:
Answer:
types 4,8,10
Explanation:
1.
Fibril-forming collagens (I, II, III, V, XI, XXIV, XXVII);
2.
Fibril-associated collagens with interrupted triple helices (FACITs) (IX, XII, XIV, XVI, XIX, XX, XXI, XXII). The FACITs do not form fibrils by themselves but they are associated with the surface of collagen fibrils.
3.
Network-forming collagens (IV, VIII, X) form a pattern in which four molecules assemble via their amino-terminal 7S domain to form tetramers while two molecules assemble via their carboxy-terminal NC1 domain to form NC1 dimers
4.
Membrane collagens (XIII, XVII, XXIII, XXV)
