Answer:
No precipitate is formed.
Explanation:
Hello,
In this case, given the dissociation reaction of magnesium fluoride:
And the undergoing chemical reaction:
We need to compute the yielded moles of magnesium fluoride, but first we need to identify the limiting reactant for which we compute the available moles of magnesium chloride:
Next, the moles of magnesium chloride consumed by the sodium fluoride:
Thus, less moles are consumed by the NaF, for which the moles of formed magnesium fluoride are:
Next, since the magnesium fluoride to magnesium and fluoride ions is in a 1:1 and 1:2 molar ratio, the concentrations of such ions are:
![[Mg^{2+}]=\frac{3x10^{-4}molMg^{+2}}{(0.3+0.5)L} =3.75x10^{-4}M](https://tex.z-dn.net/?f=%5BMg%5E%7B2%2B%7D%5D%3D%5Cfrac%7B3x10%5E%7B-4%7DmolMg%5E%7B%2B2%7D%7D%7B%280.3%2B0.5%29L%7D%20%3D3.75x10%5E%7B-4%7DM)
![[F^-]=\frac{2*3x10^{-4}molMg^{+2}}{(0.3+0.5)L} =7.5x10^{-4}M](https://tex.z-dn.net/?f=%5BF%5E-%5D%3D%5Cfrac%7B2%2A3x10%5E%7B-4%7DmolMg%5E%7B%2B2%7D%7D%7B%280.3%2B0.5%29L%7D%20%3D7.5x10%5E%7B-4%7DM)
Thereby, the reaction quotient is:
In such a way, since Q<Ksp we say that the ions tend to be formed, so no precipitate is formed.
Regards.
The balanced chemical reaction is written as:
<span>NaOH + HCl → NaCl + H2O
We are given the amount of sodium hydroxide to be used up in the reaction. This will be the starting point for the calculation.
2.75 x 10^-4 mol NaOH ( 1 mol H2O / 1 mol NaOH ) ( 18.02 g H2O / 1 mol H2O ) = 4.96 x 10^-3 g H2O</span>
Answer:hope we can be friends
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Although phlorizin inhibition of Na+-glucose cotransport occurs within a few seconds, 3H-phlorizin binding to the sodium-coupled glucose transport protein(s) requires several minutes to reach equilibrium (the fast-acting slow-binding paradigm). Using kinetic models of arbitrary dimension that can be reduced to a two-state diagram according to Cha’s formalism, we show that three basic mechanisms of inhibitor binding can be identified whereby the inhibitor binding step either (A) represents, (B) precedes, or (C) follows the rate-limiting step in a binding reaction. We demonstrate that each of mechanisms A–C is associated with a set of unique kinetic properties, and that the time scale over which one may expect to observe mechanism C is conditioned by the turnover number of the catalytic cycle. In contrast, mechanisms A and B may be relevant to either fast-acting or slow-binding inhibitors.
Explanation:
Take the L an Ricky rolled
