Hypertonic environment
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How do salts and sugars preserve food?</h3>
Salts and sugars work to preserve foods by creating a hypertonic environment. Salt and sugar will remove the water from the bacteria or fungi and they will not be able to proliferate. Loss of water results in plasmolysis, or cytoplasmic shrinkage.
<h3>What is hypertonic solution and plasmolysis?</h3>
Compared to another solution, a hypertonic solution has a higher solute concentration.
Plant cells subjected to hyperosmotic stress frequently exhibit plasmolysis as a reaction. The live protoplast violently separates from the cell wall as a result of the loss of turgor. The vacuole is primarily responsible for the plasmolytic process.
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it is grasshopper because they donot have backbones
Answer:A. Water moves into the cell
Explanation:water moves into the cell through osmosis.during osmosis water moves from a region of low concentration of solute to a region of high concentration of solute.the glucose introduced into the cell makes it more concentrated.
In this case the cell is hypertonic and water would enter into the cell through the semi permeable membrane.this membrane allows water to pass through but not glucose.this movement of water into the cell causes the cell to become turgid.
Capsid is the component of a virus that is lacking in its cell
A capsid is the protein shell that surrounds a virus. Capsids functions by protecting the nucleic acids of a virus while interacting with the host environment. Capsids are made up of many oligomeric structural subunits that contains the protein; protomers. Capsids are generally grouped based on their structure. The common ones are helical and icosahedral.
<span>There are numerous proteins in muscle. The main two are thin actin filaments and thick myosin filaments. Thin filaments form a scaffold that thick filaments crawl up. There are many regulatory proteins such as troponin I, troponin C, and tropomyosin. There are also proteins that stabilize the cells and anchor the filaments to other cellular structures. A prime example of this is dystrophin. This protein is thought to stabilize the cell membrane during contraction and prevent it from breaking. Those who lack completely lack dystrophin have a disorder known as Duchene muscular dystrophy. This disease is characterized by muscle wasting begininng in at a young age and usually results in death by the mid 20s. The sarcomere is the repeating unit of skeletal muscle.
Muscle cells contract by interactions of myosin heads on thick filament with actin monomers on thin filament. The myosin heads bind tightly to actin monomers until ATP binds to the myosin. This causes the release of the myosin head, which subsequently swings foward and associates with an actin monomer further up the thin filament. Hydrolysis and of ATP and the release of ADP and a phosphate allows the mysosin head to pull the thick filament up the thin filament. There are roughly 500 myosin heads on each thick filament and when they repeatedly move up the thin filament, the muscle contracts. There are many regulatory proteins of this contraction. For example, troponin I, troponin C, and tropomyosin form a regulatory switch that blocks myosin heads from binding to actin monomers until a nerve impulse stimulates an influx of calcium. This causes the switch to allow the myosin to bind to the actin and allows the muscle to contract. </span><span>
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