Answer:
Explanation:
Cellular respiration generally involves breaking down of large organic molecules to release ATP (energy). Citric Acid cycle, also known as Kreb's cycle or Tricarboxylic acid cycle is the second stage of the cellular respiration (unique to aerobic organisms). Citric acid cycle occurs in the intracellular space or matrix of the mitochondria of eukaryotes.
Glycolysis, which is the first step of cellular respiration, produces pyruvate which is then converted to Acetyl CoA in order to enter the Kreb's cycle by first combining with oxaloacetate. Generally, citric acid cycle involves an eight-steps reaction consisting of series of reduction-oxidation, hydration, dehydration, decarboxylation reactions, with each step catalyzed by different enzymes.
In a nutshell, oxaloacetate is generated back at the completion of the cycle alongside 2 molecules of CO2, one GTP/ATP molecule and electron donors; NADH2 and FADH2. These reduced electron donors enter the third step of aerobic cellular respiration and act as the first electron donor in the Electron transport chain.
Answer:
rap is about that life but pop is different it is not about tje life of a gangster
Explanation:
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Correct Completed Questions; Susan, a 67 year old female is experiencing ataxia, or poor muscle coordination. Doctors perform a neurological exam and they find that Susan is not positive for the Romberg test. Describe what these findings suggest about the sources of Susan's ataxia.
Answer:s
In order to test for balance and positioning(proprioception) as a measure of neurological function, or to test level of intoxication alcohol during driving, Romberg test is usually conducted,which can be negative or positive.It is generally used to investigate the cause of loss of motor control in individuals.
Explanation:
A positive result showed that her ataxia was due to loss of positioning due to loss of sensory from sensory neurons.However if negative as in the case of Susan, then it is to due to abnormality/dysfunctions in the cerebellum, and not due to loss of sensory functions,
Answer:
Indivisuals with producing an abnormal form of extracellular protein fibrillin are suffering from Marfan syndrome ehich is caused by genetic mutation in the FBN1 gene.
Explanation:
Gene mutations in FBN1 gene results in the production of an abnormal extracellular matrix fibrillin-1 protein that cannot function properly. These gene mutations basically reduce the amount of fibrillin-1 produced by the cell, alter the structure of fibrillin-1, or causes the impairment of the transport of fibrillin-1 out of the cell.
As a result, protein is poorly incorporated into extracellular matrix. Hence, indivisuals with Marfan syndrome present following symptoms
Tall stature.
Disproportionately long arms, legs and fingers.
Sternum either protrudes outward or dips inward.
Arched palate and crowded teeth.
Heart murmurs.
Extreme nearsightedness.
