Answer:
A. Inhibit FSH secretion.
Explanation:
Inhibin is a hormone secreted by the granulosa cells in the ovaries of women and the Sertolli cells in men. The main function of inhibin, as they name says, is to inhibit the secretion of FSH by the anterior pituitary gland. Inhibin is also produced by the Sertolli cells in the testes of men, and it is released in the blood when the sperm count is too high. FSH causes the Sertoli cells of the testes to begin the process of spermatogenesis in the testes. Therefore, releasing inhibin would cause a negative feedback and stop the production of sperm.
Adolescents differ from adults in the way they behave, solve problems, and make decisions. Other changes in the brain during adolescence include a rapid increase in the connections between the brain cells and making the brain pathways more effective.
Answer:
opioid receptors in the brain.
Explanation:
High-fructose corn syrup, sorbitol and sucrose are the nutritive sweeteners.
Nutritive sweeteners, conjointly referred to as caloric sweeteners or sugars, give energy within the style of carbohydrates. Non-nutritive sweeteners, conjointly known as sugar substitutes or artificial sweeteners, are alternatives that contain zero or terribly low amounts of carbohydrates or energy.
Sorbitol is a form of carbohydrate known as a sugar alcohol, or polyol. Sorbitol contains regarding third fewer calories than sugar and is sixty % as sweet. Sorbitol occurs naturally in a very sort of berries and fruits (e.g., apples and blackberries).
Ingestion an excessive amount of high-fructose corn syrup may promote weight gain, and neither sweetener has any wholesome worth on the far side the calories.
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Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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