<span>A drug used to treat CML, imatinib, binds to the active site of Abl kinase. Why does this drug work to treat this type of cancer?
</span><span>B) By binding to the active site, the drug prevents the ability of Abl kinase to bind to its substrate.
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Imatinib works against CML by binding close to the ATP binding site of bcr-abl. The binding results to the<span> locking in of the bcr-abl to a closed or self-inhibited conformation and inhibiting the enzyme activity of the protein </span><span>semi-competitively.</span>
<span>I belive the answer is D. Burbank's process did not involve environmental pressures to shape the outcome.
Burbank process is similar to natural selection because he will select the potato based on some traits. The trait would be big potato and resistant to disease. The genetic makeup will also favor those with the selected trait. This process should be faster.</span>
The virus' DNA becomes a part of the host cell's DNA, and every time the host cell copies and divides, it also copies viral DNA. The viral DNA may remain inactive (a provirus) for a long time, but it can become active when it frees itself from the host's chromosome, which triggers the lytic cycle.
I forget which one is the virus' DNA
