<span>When you are pregnant, you do not ovulate because you have already fertilized an egg and it is embedded into the uterus where it is growing into a baby. The body knows this, so it does not shed the uterine lining. The shedding of this lining is triggered by hormones which change when the body is pregnant. During pregnancy, a large amount of progesterone is initially produced which helps to prevent the lining from shedding. If you are not pregnant, your progesterone level will begin to decline at the end of your cycle, triggering the shedding of the lining and the period. Birth control pills alter your hormones to mimic a pregnant state. This will prevent the body from releasing an egg and then shedding the uterine lining.</span>
Answer:
Correct answer is ''c'' Emergency Medical Dispatchers
Explanation:
A 911 emergency medical dispatcher is a professional communicator who is trained to gather information related to medical emergencies, they must provide assistance and instructions by voice, prior to the arrival of the (EMS) regularly they use a series of key questions, pre-arrival instructions, and dispatch priorities to be used in the processing of EMS calls.
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
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Answer:
tRNA
Explanation:
transfer RNA (tRNA) assembles the protein (one amino acid at a time)
