Answer: Incomplete dominance
Explanation: Incomplete dominance is a type of inheritance, specifically a type of intermediate inheritance when a dominant allele, or form of a gene, does not completely mask the effects of a recessive allele, and the organism’s resulting physical appearance shows a blending of both alleles. The result is a phenotype (expression) where the expressed physical trait is a combination of both of the phenotypes that belong to the alleles. One allele doesn’t mask or dominate the other alleles in this instance. It is also called semi-dominance or partial dominance.
In short, incomplete dominance is when neither gene is fully dominant, and the result is a brand new trait.
The Punnett square shows genetic inheritance as a simple model with only two different versions of alleles: dominant and recessive. In this simple relationship, dominant alleles always override the recessive alleles to be expressed in the organism’s appearance or phenotype. It was created by Gregor Mendel and was important because it contradicted popular ideas at the time that the traits of the parents were simply permanently blended within their offspring. However, modern biologists have discovered that inheritance isn’t as simple as this model would suggest.
An example of incomlete dominance in humans would be hypercholesterolemia.
Most likely true because for all living things, their DNA is constantly mutating to keep itself alive in a certain environment,
Answer:
Fats would accumulate in the liver and the individual would show hypocholesterolemia
Explanation:
Familial hypercholesterolemia is a genetic disorder caused by a reduction and/or defect in the low-density lipoprotein (LDL) receptor. Moreover, apolipoprotein B is the unique protein component of LDL, which plays a critical role in lipid metabolism by binding the LDL receptor. Familial defective apolipoprotein B-100 refers to an inherited disorder associated with mutations of the apolipoprotein B-100 that leads to moderate or severe cases of hypercholesterolemia. Mutations in apolipoprotein B-100 are associated with a reduction in the binding capacity of LDL to the LDL receptor, thereby leading to hypercholesterolemia.
Answer:
Feedback Inhibition
Explanation:
Yes, this mechanism being one of importants for the regulatory mechanism in our cells. They use non competetive inhibitors. Example: Synthesis of amino acids, etc.
Answer:
D if I'm D correct, or A maybe C, mostly D.
