<h2>it carries oxygen from the lungs to rest of the body.</h2><h2>This is a fast function and i hope it helps...</h2>
Answer:
Hepato- and nephrotoxicity of fluoride have been demonstrated in animals, but few studies have examined potential effects in humans. This population-based study examines the relationship between chronic low-level fluoride exposure and kidney and liver function among United States (U.S.) adolescents. This study aimed to evaluate whether greater fluoride exposure is associated with altered kidney and liver parameters among U.S. youth.
This cross-sectional study utilized data from the National Health and Nutrition Examination Survey (2013–2016). We analyzed data from 1983 and 1742 adolescents who had plasma and water fluoride measures respectively and did not have kidney disease. Fluoride was measured in plasma and household tap water. Kidney parameters included estimated glomerular filtration rate (calculated by the original Schwartz formula), serum uric acid, and the urinary albumin to creatinine ratio. Liver parameters were assessed in serum and included alanine aminotransferase, aspartate aminotransferase, alkaline phosphatase, blood urea nitrogen, gamma-glutamyl transferase, and albumin. Survey-weighted linear regression examined relationships between fluoride exposure and kidney and liver parameters after covariate adjustment. A Holm-Bonferroni correction accounted for multiple comparisons.
The average age of adolescents was 15.4 years. Median water and plasma fluoride concentrations were 0.48 mg/L and 0.33 μmol/L respectively. A 1 μmol/L increase in plasma fluoride was associated with a 10.36 mL/min/1.73 m2 lower estimated glomerular filtration rate (95% CI: −17.50, −3.22; p = 0.05), a 0.29 mg/dL higher serum uric acid concentration (95% CI: 0.09, 0.50; p = 0.05), and a 1.29 mg/dL lower blood urea nitrogen concentration (95%CI: −1.87, −0.70; p < 0.001). A 1 mg/L increase in water fluoride was associated with a 0.93 mg/dL lower blood urea nitrogen concentration (95% CI: −1.44, −0.42; p = 0.007).
Fluoride exposure may contribute to complex changes in kidney and liver related parameters among U.S. adolescents. As the study is cross-sectional, reverse causality cannot be ruled out; therefore, altered kidney and/or liver function may impact bodily fluoride absorption and metabolic processes.
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This is likely a mutant mouse (as synaptic clefts are typically 10 to 20 nm wide). Given that neurotransmitters act over short distances, this mouse may demonstrate delayed responses to stimuli, or no responses at all.
That’s the answer
Carbon dioxide and water
<span>C6</span><span>H12</span><span>O6</span>+<span>O2</span>→<span>CO2</span>+<span>H2</span>O+<span>energy</span>
Answer:
One of the factors for the development of pyelonephritis, in addition to E. coli, is urinary retention and reflux (option a).
Explanation:
Pyelonephritis is an infection of the upper urinary tract caused by bacteria in the urine, such as Escherichia coli.
Under normal conditions, urine in the urinary bladder is aseptic, that is, without bacteria. The presence of bacteria in the urine indicates a urinary infection.
Urinary retention is the limitation of the expulsion of urine from the bladder. This promotes:
- <em>An increase in the amount of bacteria present in the bladder.
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- <em>The pressure generated by urine retention causes the bladder to generate a retrograde flow - reflow - towards the ureters, leading the bacteria to the kidneys.
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The result of urinary retention and reflux - when bacteria are present - is an infection in the upper urinary tract, called pyelonephritis.
Learn more:
Urinary tract infection brainly.com/question/4756206
