The beneficial uses of bacterial toxins in medicine are more and more used lately. For example, Botulinum toxin is a toxic protein produced by the bacterium Clostridium botulinum which has paralytic effects (injection of this toxin into muscle relax specific muscles). Botulinum toxin accomplishes his effects on the neuromuscular junction where he prevents the release of the neurotransmitter acetylcholine (Ach). Utilization of this toxin is in the treatment of various muscle spasms. It is also used in the treatment of migraines. Diphtheria toxin is also one of the toxins used for medical purposes for the treatment of cutaneous and non-Hodgkin T-cell lymphomas. <span>Some bacterial toxins can be used in the treatment of tumours. For example, immunotoxin, which is protein made by fusion of modified antibody and toxin.The antibody binds to an antigen on the target cell, the toxin then enters via endocytosis and kills the cell. Commonly used bacterial toxins in immunotoxins are Diphtheria toxin and the Pseudomonas exotoxin.</span>
Explanation:
Mosses go through sporophyte and gametophyte stages in their life cycle. ... However, mosses are nonvascular while ferns are vascular. Gymnosperms and angiosperms are both vascular, seed-bearing plants. However, gymnosperms release their seeds in cones (like pinecones) while angiosperms release their seeds through flowers.
a cell adapts using natural celection in the long term
in the short term, look at this Wikipedia article.
https://en.wikipedia.org/wiki/Cellular_adaptation
<span>There are numerous proteins in muscle. The main two are thin actin filaments and thick myosin filaments. Thin filaments form a scaffold that thick filaments crawl up. There are many regulatory proteins such as troponin I, troponin C, and tropomyosin. There are also proteins that stabilize the cells and anchor the filaments to other cellular structures. A prime example of this is dystrophin. This protein is thought to stabilize the cell membrane during contraction and prevent it from breaking. Those who lack completely lack dystrophin have a disorder known as Duchene muscular dystrophy. This disease is characterized by muscle wasting begininng in at a young age and usually results in death by the mid 20s. The sarcomere is the repeating unit of skeletal muscle.
Muscle cells contract by interactions of myosin heads on thick filament with actin monomers on thin filament. The myosin heads bind tightly to actin monomers until ATP binds to the myosin. This causes the release of the myosin head, which subsequently swings foward and associates with an actin monomer further up the thin filament. Hydrolysis and of ATP and the release of ADP and a phosphate allows the mysosin head to pull the thick filament up the thin filament. There are roughly 500 myosin heads on each thick filament and when they repeatedly move up the thin filament, the muscle contracts. There are many regulatory proteins of this contraction. For example, troponin I, troponin C, and tropomyosin form a regulatory switch that blocks myosin heads from binding to actin monomers until a nerve impulse stimulates an influx of calcium. This causes the switch to allow the myosin to bind to the actin and allows the muscle to contract. </span><span>
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Answer:
An arterie is a vessel that carries blood away from the heart, and a vein carries blood to the heart
Explanation:
Mark as barinly plzzz :))))