A recent meta-analysis of seven studies completed that dietary intakes (not vitamin supplements) of vitamin E, C, and beta-carotene existed linked to a decreased risk of Alzheimer disease.
<h3>What is the Causes of Alzheimer?</h3>
In individuals with early-onset Alzheimer's, a genetic mutation may be the cause. Late-onset Alzheimer's arises from a complicated series of brain changes that may happen over decades. The causes probably contain a combination of genetic, environmental, and lifestyle factors.
Symptoms may include:
- Increased memory loss and disarray.
- Inability to discover unique things.
- Problem with language and problems with reading, writing, and performing with numbers.
- Difficulty managing thoughts and thinking logically.
- Compressed attention span.
- Problems managing new situations.
There's currently no treatment for Alzheimer's disease. But there exists medicine available that can temporarily lower the symptoms. Support stands also available to assist someone with the condition, and their family, coping with everyday life.
Hence, A recent meta-analysis of seven studies completed that dietary intakes (not vitamin supplements) of vitamin E, C, and beta-carotene existed linked to a decreased risk of Alzheimer disease.
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Answer:
Small diameter nerves are the first to be anesthetized. Myelinated nerves are blocked faster than unmyelinated nerves. Because size is thought to outweigh myelination, nerve fibers are blocked
Explanation:
She should gain between 25-35 pounds. Based on her BMI she should weight approx 173 lbs
Answer:
b) blastic red blood cell (RBC).
Explanation:
In excess of 340 blood group antigens have now been described that vary between individuals. Thus, any unit of blood that is nonautologous represents a significant dose of alloantigen. Most blood group antigens are proteins, which differ by a single amino acid between donors and recipients. Approximately 1 out of every 70 individuals are transfused each year (in the United States alone), which leads to antibody responses to red blood cell <u>(RBC) alloantigens</u> in some transfusion recipients. When alloantibodies are formed, in many cases, RBCs expressing the antigen in question can no longer be safely transfused. However, despite chronic transfusion, only 3% to 10% of recipients (in general) mount an alloantibody response. In some disease states, rates of alloimmunization are much higher (eg, sickle cell disease). For patients who become alloimmunized to multiple antigens, ongoing transfusion therapy becomes increasingly difficult or, in some cases, impossible. While alloantibodies are the ultimate immune effector of humoral alloimmunization, the cellular underpinnings of the immune system that lead to ultimate alloantibody production are complex, including antigen consumption, antigen processing, antigen presentation, T-cell biology.
Answer:
True
Explanation:
The CDC posted a report which states these facts. Sadly, people do not engaged in as much physical activity as they should.
