Answer:
pulmonary disease.
Explanation:
with this condition, diaphragm has symptoms of not inhaling enough o2, absorbing the o2 into the bloodstream, caused by smoking, COPD, etc. results in hypo ventilation, and heart conditions. diaphragm is what helps lungs inhale & exhale o2 & co2...
Answer:
As, this individual is expending fructose syrup.
Fructose is changed over to purine which is additionally changed over to uric acid.
Along these lines, more fructose in the eating routine then more purines in the body which will additionally frame progressively uric acid.
This uric acid gathers and causes gout.
What is the best clarification for the introduction of gout right now please clarify why different decisions are not reliable with the information or what extra data you may need to choose the underlying driver?
1. overproduction of purines-They are not being created in the body however yes they are certainly are delivered from the abundance fructose expended.
2. diminished rescue of purines-No, this isn't the main driver.
3. diminished urinary discharge of uric acid this can occur as less uric acid is being discharged in the pee.
When, contrasted with the ordinary individual the patient is discharging less uric acid.
Along these lines, third point is the purpose behind gathering of uric acid in the body.
Answer:
d is correct am pretty sure
Explanation:
hope it helps
Huh lol? do you have a question or something
Answer:
types 4,8,10
Explanation:
1.
Fibril-forming collagens (I, II, III, V, XI, XXIV, XXVII);
2.
Fibril-associated collagens with interrupted triple helices (FACITs) (IX, XII, XIV, XVI, XIX, XX, XXI, XXII). The FACITs do not form fibrils by themselves but they are associated with the surface of collagen fibrils.
3.
Network-forming collagens (IV, VIII, X) form a pattern in which four molecules assemble via their amino-terminal 7S domain to form tetramers while two molecules assemble via their carboxy-terminal NC1 domain to form NC1 dimers
4.
Membrane collagens (XIII, XVII, XXIII, XXV)