Claim: the mean resting pulse rate for men is less than 72
bpm.
Symbolic form would be: μ < 72
Step 1: null hypothesis : μ > = 7 2 b p m
Step 2: alternative hypothesis: μ < 7 2 b p m
Step 3: the test statistic is z = mean – μ / standard dev/sqrt (n) = -3/4 / 1.7639 = -1.92
Step 4: c v method: since z α = -1.645, the test static is z = -1.92 is less than the CV, we reject null hypothesis
P Value method: area is 0.0274, since the p value is <= α, reject null hypothesis.
Step 5: the sample data support the claim that the mean resting pulse rate for men is less than 72 bpm.
Answer:
Plant cells have a large central vacuole, a cell wall, and chloroplasts while animal cells do not.
Hope this helps!
Answer:
Central posterolateral thalamus.
Explanation:
The third order neuron originates from Central posterolateral nuclei thalamus because it is the nucleus of thalamus because it receive impulses from the neospinothalamic tract and the medial lemniscus of the posterior column-medial lemniscus pathway i.e it received sensory information from the body, send it to the brain and then transfer the information to brodman.
Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
