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Gre4nikov [31]
2 years ago
12

After cell division (mitosis) how does the genetic code of the newly

Biology
1 answer:
Harrizon [31]2 years ago
3 0

Answer:

Chromosomes were first named by cytologists viewing dividing cells through a microscope. The modern definition of a chromosome now includes the function of heredity and the chemical composition. A chromosome is a DNA molecule that carries all or part of the hereditary information of an organism. In eukaryotic cells, the DNA is packaged with proteins in the nucleus, and varies in structure and appearance at different parts of the cell cycle.

Explanation:

Cells reproduce genetically identical copies of themselves by cycles of cell growth and division. The cell cycle diagram on the left shows that a cell division cycle consists of 4 stages:

G1 is the period after cell division, and before the start of DNA replication. Cells grow and monitor their environment to determine whether they should initiate another round of cell division.

S is the period of DNA synthesis, where cells replicate their chromosomes.

G2 is the period between the end of DNA replication and the start of cell division. Cells check to make sure DNA replication has successfully completed, and make any necessary repairs.

M is the actual period of cell division, consisting of prophase, metaphase, anaphase, telophase, and cytokinesis.

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If I found the same gene in all organisms that I've tested, I would be intrigued because that would be a giant step in evolution. My reason for this answer is because if you have the same gene that would technically mean we all specifically came from the same species of animals.

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3 years ago
Need help with biology
padilas [110]

Answer

Hi,

A. Radioactive markers

Explanation

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Answer:

the answer is A. E. coli B

Explanation:

The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.

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