If you want to grow up a large quantity of streptomycin-resistant E. coli, you would require to pick a colony of the bacteria from the streptomycin-positive plate and allow to grow it on a streptomycin positive plate.
<h3>What is E. coli?</h3>
E. coli may be defined as a type of bacterium that is commonly present in the intestinal regions of humans and other animals, some strains of this bacterium can significantly cause severe food poisoning.
The strain of streptomycin-positive is those population of E. Coli which is significantly streptomycin resistant, while the negative strain has the opposite effect.
That's why if you want to grow up a large quantity of streptomycin-resistant, you must remarkably require to pick only a positive strain of streptomycin for E.Coli bacterium.
Therefore, if you want to grow up a large quantity of streptomycin-resistant E. coli, you would require to pick a colony of the bacteria from the streptomycin-positive plate and allow to grow it on a streptomycin-positive plate.
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Answer:
First Law of Thermodynamics in Biological Systems
All biological organisms require energy to survive. ... The chemical energy is stored in the form of glucose, which is used to form complex carbohydrates necessary to build plant mass. The energy stored in glucose can also be released through cellular respiration.
Explanation:
Answer:
Fungi have chitin as a component of their cell walls rather than cellulose, which is one of the primary differences between plants and fungi. Chitin and cellulose are both made up of polysaccharide chains. Another difference between plants and fungi is that plants contain chlorophyll whereas fungi do not.
Answer:
Aspirin works by inhibiting the production of prostaglandins. Aspirin inhibits the formation of prostaglandins by combining with the COX enzymes. Prostaglandins function as messenger molecules to monitor different physiological procedures in distinct regions of the body. One of the prime activities of prostaglandins is to stimulate inflammation and pain.
Prostaglandins are also the essential controller of platelet aggregation. By changing the COX enzymes inside the platelets, aspirin makes platelets to lose the stickiness, which is required to instigate clotting of blood.
There are two forms of cyclooxygenase, that is, COX-1 and COX-2. COX-1 generates prostaglandins and COX-2 mediates pain and swelling in response to tissue injury. Aspirin prevents both COX-1 and COX-2 functioning, while COX-2 is the therapeutic target of the drug.
However, it is the association of aspirin with COX-1 in the gastrointestinal tract, which results in the unwanted side effects of the drug. COX-1 is required to sustain a thick lining of the stomach. As aspirin inhibits the COX-1 enzyme, thus, the continuous use of the drug can result in the thinning of mucus, which safeguards the stomach from gastric juices.
In such cases, stomach bleeding, ulcers, and in certain situations perforation of the stomach can take place. Therefore, aspirin exhibits both bad and good effects.
