Answer:
Electron Transport Chain
In the third stage, electrons from the NADH and FADH2 molecules move to the electron transport chain (ETC). These electrons generate the required power to move protons through the mitochondria into the space between membranes. In the final stage of cellular respiration, oxygen combines with the electrons and protons to form water.
Answer:
knowledge of the sequence product
Explanation:
A gene knockout is a technique used in molecular genetics to deactivate target genes in an organism in order to study their functions by reverse genetics (i.e., gene loss). Knockouts are generated by different methods including, for example, homologous recombination or site-specific nucleases (zinc-fingers, TALENS, CRISPR/Cas9). These techniques require to know a priori the sequence of each gene to be knocked out in order to target desired mutations. In the last years, the CRISPR/Cas9 tool has gained attention to knockout genes of interest because it is a genome editing system that can be easily used for deletion or insertion of bases.
Answer:
Roughly 96 percent of the mass of the human body is made up of just four elements: oxygen, carbon, hydrogen and nitrogen, with a lot of that in the form of water. ... Sulfur is not usually mentioned as a dietary supplement because the body gets plenty of it in proteins. Humans get nitrogen from food. The element is an important component of amino acids, which are used to build peptides and proteins. Nitrogen is also an essential component of the nucleic acids DNA and RNA and all of the other molecules derived from the nitrogenous bases.
Explanation:
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Leukocytes are produced/stored in thymus, spleen, and bone marrow
He demonstrated that SCD and sickle cell trait were due to the presence of abnormal 8-globin polypeptides in red blood cells. He demonstrated that the electrophhoretic mobility of B-globin from patients with SCD was different from that of healthy individuals. He demonstrated that both parents of multiple patients with SCD had low levels of sickled red blood cells. He hypothesized that SCD was a recessive trait and that the parents of patients with SCD would be heterozygous carriers. He demonstrated that the difference between B-globin polypeptides in individuals who were healthy and those with SCD is an amino acid substitution. He performed a peptide fingerprint analysis on B-globin from individuals with 84 84 and 89 88, which identified the segment of B-globin that was changed by the BS mutation. James Neel Linus Pauling Vernon Ingram
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