Specializations of the small intestine are related to surface absorption it doesn't include Peyer's patches.
<h3>What is the small intestine?</h3>
It lies between the stomach and the large intestine and is by far the longest part of the digestive tract.
It presents various specializations that guarantee an increase in the surface area for absorption of nutrients such as circular folds, microvilli, villi.
Therefore, we can conclude that specializations of the small intestine are related to surface absorption it doesn't include Peyer's patches.
Learn more about the small intestine here: brainly.com/question/11348399
Answer:
Monotremes, marsupials, and placentals are the three animals that could match this phylogenetic tree.
Explanation:
Monotremes, marsupials, and placentals belong from a common ancestor. Those organisms which have one common ancestor are also called monophyletic. These animals have some changes which occurs with the passage of time but most of characters are similar to each other. In placentals animals placenta is present while in Monotremes and marsupials this placenta is absent.
Answer:Acoustic Neuroma (Vestibular Schwannoma)
Explanation:An acoustic neuroma is a noncancerous brain tumor that develops on the nerve that runs between the inner ear and the brain. Also called vestibular schwannomas, acoustic neuromas develop in cells known as Schwann cells. The primary symptoms of vestibular schwannoma are unexplained progressive unilateral hearing loss and tinnitus(the perception of sound when no external corresponding sound is present) and vestibular (disequilibrium) symptoms.
Answer:
Id say C. developmental psychologist
Explanation:
Answer:
Thanks for you question. Your hypothesis suggests a linear relationship between serum Cholesterol levels and MI. This hypothesis seems to ignore the difference in the prevalence and effectiveness of LDL receptors in the FH patient.
FH patients who have inherited the mutation from both parents have very few LDL receptors in their blood and therefore almost no ability to pass the unused Cholesterol through the liver. FH patients who are heterozygous will have more LDL receptors although both will find Cholesterol removal problematic without the addition of a PCSK9 inhibitor.
In short, your hypothesis need to account for other factors that are in play.
Explanation:
Consider my case. I am a 64 year old male who has Heterozygous Familial Hypercholesterolemia. Before treatment at age 12 my Total cholesterol was 510 mg/dl. My genetic testing shows two mutations to the LDL Receptor gene with only one mutation being pathogenic. My first heart attack was at 47 and first stroke at 62. My current LDL is too low to detect with the use of a PCSK9 inhibitor (Repatha®).
