Odorants can stimulate somatosensory system through polymodal nociceptors (touch, pain, temperature receptors), these sensations are mediated by the trigeminal nerve (cranial nerve), which transmits information about the feel of an odorant. Often, it is impossible to distinguish between sensations traveling up cranial nerve I from olfactory receptors and those traveling up cranial nerve V from somatosensory receptors e.g. peppermint, cool, Amonia, burning, etc.
Hi there,
This would be called mutation
<span>Conducting an experiment is the step in the
scientific method after making a prediction.
hope this helps, UnbotheredMaariee!! :)</span>
<span>The appendicular skeleton includes the bones of the shoulder girdle, the upper limbs, the pelvic girdle, and the lower limbs.</span>
The question is incomplete. The complete question is:
Motor neurons release the neurotransmitter acetylcholine (ACh) and acetylcholinesterase degrades ACh in the synapse. If a neurophysiologist applies onchidal (a naturally occurring acetylcholinesterase inhibitor produced by the mollusk Onchidella binneyi) to a synapse, what would you expect to happen?
A) paralysis of muscle tissue
B) convulsions due to constant muscle stimulation
C) decrease in the frequency of action potentials
D) no effect
Answer:
B. convulsions due to constant muscle stimulation
Explanation:
In response to an action potential, acetylcholine released at the neuromuscular junction or synapse. The release of acetylcholine is followed by its binding to the receptors in the motor endplate and finally triggering a muscle action potential which in turn leads to muscle contraction. Termination of activity of acetylcholine (Ach) is mediated by acetylcholinesterase which destroys ACh and thereby prevents firing of another muscle action potential.
If acetylcholinesterase inhibitor is administered to a synapse, acetylcholine will keep on triggering the muscle action potential causing continuous muscle contraction and irregular body movements (convulsions).