Something that isn't a major concern could be the wether because the whether usually does not effect his experimental concept. In this case his concept is testing the effects of peanut butter on short term memory
Answer:
<u> The following four traits are -: </u>
- <u>Pedigree 1 -</u> A recessive trait (autosomal recessive) is expressed by pedigree 1.
- <u>Pedigree 2- Recessive inheritance is defined by Pedigree 2. </u>
- <u>Pedigree 3</u> - The inheritance of the dominant trait (autosomal dominant) is illustrated by Pedigree 3.
- <u>Pedigree 4-</u> An X-like dominant trait is expressed by Pedigree 4.
Explanation:
<u>Explaination of each pedigree chart</u>-
- Pedigree 1 demonstrates the <u>recessive trait </u>since their children have been affected by two unaffected individuals. If the characteristics were X-linked, in order to have an affected daughter, I-1 would have to be affected.
In this, both parents are autosomal recessive trait carriers, so the child will be affected by a 1/4 (aa) - <u> Recessive inheritance</u> is defined by <u>Pedigree 2</u>. This is<u> X-related inheritance as autosomal recessive</u> inheritance has already been accounted for in part 1. This inference is confirmed by evidence showing that the father (I-1) is unaffected and that only the sons exhibit the characteristic in generation II, suggesting that the mother must be the carrier. The individual I-2 is a carrier for this X-linked trait. A typical Xa chromosome is attached to the unaffected father (I-1), so the chance of carrier II-5 is 1/2. Probability of an affected son = 1/2 (probability II-5 is a carrier) x 1/2 (probability II -5 contributes (
) x 1/2 (probability of Y from father II-6) = 1/8. An affected daughter's likelihood is 0 because a typical
must be contributed by II-6. - The inheritance of the<u> dominant trait</u> is demonstrated by <u>Pedigree 3 </u>because affected children still have affected parents (remember that all four diseases are rare). The trait must be <u>autosomal dominant</u> because it is passed down to the son by the affected father. There is a 1/2 risk that the heterozygous mother (II-5) would pass on mutant alleles to a child of either sex for an autosomal dominant feature.
- <u>Pedigree 4</u> is an <u>X-linked dominant function</u> characterized by the transmission to all of his daughters from the affected father but none of his son. On the mutant X chromosome, the father (I-1) passes on to all his daughters and none of his sons. As seen by his normal phenotype, II-6 therefore does not bear the mutation. An affected child's likelihood is 0.
In the question the pedigree chart was missing ,hence it is given below.
Answer:
The cell membrane, also called the plasma membrane, is found in all cells and separates the interior of the cell from the outside environment. The cell membrane consists of a lipid bilayer that is semipermeable. The cell membrane regulates the transport of materials entering and exiting the cell.
Explanation:
<span>The organic molecules that must be included in the soil for earthworm nutrition are Neutral pH. A neutral pH substance that is neither basic nor acidic is considered neutral. The pH scale is the one that measures how a substance is basic or acidic. It ranges from 0-14. A 7 pH is neutral.</span>
A mutation within a gene that will insert a untimely cease codon in mRNA would result in a shortened polypeptide chain.
<h3>What occurs if there is a untimely end codon?</h3>
Thus, nonsense mutations occur when a premature nonsense or end codon is added in the DNA sequence. When the mutated sequence is translated into a protein, the resulting protein is incomplete and shorter than normal. Consequently, most nonsense mutations result in nonfunctional proteins
<h3>What mutation motives untimely cease codon?</h3>
In genetics, a nonsense mutation is a factor mutation in a sequence of DNA that effects in a premature stop codon, or a nonsense codon in the transcribed mRNA, and in a truncated, incomplete, and normally nonfunctional protein product.
Learn more about mutation here:
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