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mafiozo [28]
3 years ago
7

MASTERY CONNECT MITOSIS

Biology
1 answer:
puteri [66]3 years ago
8 0
First one is the answer
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Question 1 (2 points)
solniwko [45]

Answer:

DNA

Explanation:

RNA is not a double helix

Uracil is on the RNA, this is a picture or DNA. Uracil is just one of the nucleic bases

This is not a monomer; it's actually a polymer

It is DNA because it is a double helix structure held together by the four nucleic bases.

7 0
3 years ago
Using jargon, or overly complex terms, can contribute to ________ noise
Illusion [34]
Answer is: <span>Using jargon, or overly complex terms, can contribute to semantic noise.
There are four forms of communication noise: </span>psychological<span> noise, physical noise, </span>physiological<span> and </span>semantic<span> noise.
</span><span>Semantic noise occurs when</span> sender of the message uses a word or a phrase (complex terms) that we don't know the meaning of.
6 0
4 years ago
What are the longest cells(when mature) in the human body?
Vinil7 [7]
I believe it is muscle cells, I'm sorry if this is incorrect.
5 0
3 years ago
What contributed to the specification in Darwin's Finch theory
Keith_Richards [23]

Answer:

On his visit to the Galapagos Islands, Charles Darwin discovered several species of finches that varied from island to island, which helped him to develop his theory of natural selection.

Darwin's finches are a classical example of an adaptive radiation. Their common ancestor arrived on the Galapagos about two million years ago. During the time that has passed the Darwin's finches have evolved into 15 recognized species differing in body size, beak shape, song and feeding behaviour.

4 0
3 years ago
The metabolic fate of fatty acids is dependent on two enzymes, carnitine acyltransferase i and acetyl-coa carboxylase. carnitine
zvonat [6]

The correct answer is the last statement.

If the regulatory serine is mutated to alanine, then acetyl-CoA carboxylase will get activated spontaneously and will produce malonyl-CoA. The increased concentrations of malonyl-CoA will obstruct the oxidation of fatty acids by preventing the entry of fatty acids into the mitochondria.

It is because the AMP-activated protein kinase phosphorylates the serine residues of acetyl-CoA carboxylase to inactivate it. If a mutation occurs in such residues, then the AMPL cannot phosphorylate acetyl-CoA carboxylase and this enzyme will get activated spontaneously.

In such a situation, there will be more than sufficient production of malonyl-CoA, which will inhibit the admittance of more fatty acid getting inside the mitochondria; this will indirectly prevent the oxidation of fatty acids.

8 0
3 years ago
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