Is called the Glottis. Hope this helps:)
Answer:
Explanation: 1) Individual who is bald but neither parents has a widow's peak: As having a peak is dominant and the individual is bald with parents that doesn't have the trait, the genotype is recessive, in the case ww;
2) Individual has a widow's peak: The individual's genotype can be WW, homozygous for the trait or Ww, heterozygous for the trait, depending on the genotype of the parents;
3) Individual can't roll their tongue: Being able to roll the tongue is a dominant characteristics, so if the individual can't roll their tongue means the genotype is recessive. But, there is little evidence that this trait is dominant or inheritable;
4) Individual has a widow's peak but their mother does not: In this case, the genotype of the individual is heterozygous (Ww), because the mother is recessive (ww) and the offspring has the trait, so the father must have had it;
5) If an individual does not have a widow's peak (ww), which genotype would be impossible? If this individual crosses over with another individual with the same trait, their children will be all recessive for widow's peak (ww), so there is no possibility of an offspring with the trait;
If the initial individual crosses over with an individual with widow's peak, two possiblities can occur:
- If the crossover is with an individual homozygous for the trait (WW): all their children will have widow's peak with genotype Ww. So, the possibility of not having the peak is 0;
- If the crossover is with an individual heterozygous for the trait (Ww), there will be a probability of 25% for the children to have genotype WW, a probability of 50% for the offspring to be heterozygous (Ww) and has the trait and probability of 25% to be recessive (ww) and therefore doesn't have the trait;
The correct answer is - B. Beak size determines what the bird can eat.
The beak of a bird can come in many different shapes and sizes. It can be pointy, long, short, rounded, scythe-like, thin, thick... All of those shapes and sizes have a specific role, and that role is to enable the bird to feed itself with certain type of food source. Every food source requires certain type of beak in order for the bird to be efficient in getting its nutrition, so depending on hat the bird eats, we can easily see a pattern in the beaks, where birds that eat nuts have one strong and shorter beak, the ones that eat warms and insects have thin, pointy one, the predator birds have claw like, sharp beak...
<span>Rhabdomyolysis constitutes a common cause of acute renal failure and presents paramount interest. A large variety of causes with different pathogenetic mechanisms can involve skeletal muscles resulting in rhabdomyolysis with or without acute renal failure. Crush syndrome, one of the most common causes of rhabdomyolysis presents increased clinical interest, particularly in areas often involved by earthquakes, such as Greece and Turkey. Drug abusers are another sensitive group of young patients prone to rhabdomyolysis, which attracts the clinical interest of a variety of medical specialties.
We herein review the evidence extracted from updated literature concerning the data related to pathogenetic mechanisms and pathophysiology as well as the management of this interesting syndrome.
Keywords: Rhabdomyolysis, acute renal failure, myoglobin, crush syndrome
The first case of the crush syndrome, which constitutes one of the main causes of rhabdomyolysis, was reported in Sicily in 1908, after an earthquake1,2. In 1930, in the Baltic area, an epidemic of myoglobinuria was observed due to consumption of contaminated fish. Interest in rhabdomyolysis and crash syndrome was stimulated during the World War II particularly after the bombing in London, where the victims developed acute renal failure and myoglobinuria1.
Rhabdomyolysis is a rupture (lysis) of skeletal muscles due to drugs, toxins, inherited disorders, infections, trauma and compression3. Lysis of muscle cells releases toxic intracellular components in the systemic circulation which leads to electrolyte disturbances, hypovolemia, metabolic acidocis, coagulation defects and acute renal failure due to myoglobin4.
The skeletal muscle consists of cylindrical myofibrils, which contain variant structural and contraction proteins. Actin and myosin, arranged in thin and thick filaments respectively, form the repeated functional units of contraction, the sarcomeres5. The sarcoplasmic reticulum constitutes an important cellular calcium storage. It is structurally connected to the t-tubules, that are formed by invaginations of the muscle cell plasma membrane, the sarcelemma, around every fibril (Figure 1). After the sarcelemma depolarization, the stimulation arrives, through the t-tubules junctions, at the sarcoplasmic reticulum, inducing the calcium ions release and triggering muscle contraction6.</span>
