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Ludmilka [50]
3 years ago
14

Read this claim from an argumentative essay about high-sugar foods. high-sugar foods should have the same sales restrictions as

other harmful substances, such as alcohol and tobacco. which piece of evidence, if true, would best support this claim?
Biology
2 answers:
swat323 years ago
8 0

Answer:

The answer is C

Explanation:

I just took the test trust me :)

Vilka [71]3 years ago
6 0
The piece of evidence that would best support this claim is; consuming too much sugar over time can lead to diabetes and cardiovascular disease. 
Diabetes is a lifestyle disease in which blood sugar or glucose levels are higher than the normal levels. Carbohydrate foods, such as breads, rice, cereals, pasta, among others can cause a rise in blood sugar. There are two types of diabetes; that is, type I and type II, type II is the one linked to high blood sugars.
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Explanation:

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3 years ago
Does the oxygen that exits photosynthesis enter as CO2? Explain.
liubo4ka [24]

Answer:

Yes

Explanation:

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irina1246 [14]

Answer:

1. P120 is degraded in the 26S proteasome

2. The 26S proteasome has a major role in protein degradation and is critical for protein homeostasis

3. Cell cycle and DNA replication are cellular processes regulated by the Ras and NFkB pathways

Explanation:

The proliferation-associated nucleolar protein (p120) is a protein known to be expressed during the interphase of the cell cycle, specifically in G1 and early S phase, where any problem with DNA replication trigger a checkpoint, i.e., a molecular cascade of signaling events that suspend DNA replication until the problem is resolved. In mammalian cells, the 26S proteasome is responsible for catalyzing protein degradation of about 80% (or even more) of their proteins. The 26S proteasome acts to degrade rapidly misfolded and regulatory proteins involved in the cell cycle, thereby having a major role in protein homeostasis and in the control of cellular processes. It is for that reason that inhibitors that block 26S proteasome function have shown to be useful as therapeutic agents in diseases associated with the failure of protein degradation mechanisms (e.g., multiple myeloma). The NF-κB are highly conserved transcription factors capable of regulating different cellular processes including, among others, cellular growth, inflammatory responses and apoptosis. Moreover, the MAPK/ERK pathway is able to transduce different signals received on the cell surface to the nucleus. The MAPK/ERK pathway is activated when a singling molecule binds to a cell receptor which triggers a signaling cascade that ends when a transcription factor induces the expression of target genes, ultimately producing a response in the cell (for example, the progression through the cell cycle).

7 0
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solong [7]
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Explanation:

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