Answer:
1. Binding of insulin to the alpha subunit of the insulin receptor
2. Activation of insulin receptor tyrosine kinase
3. Phosphorylation of IRS proteins
4. Activation of PIP3-dependent protein kinase B (PDK1)
5. Phosphorylation of phosphinositide 3-kinase (PI-3K)
6. Conversion of PIP2 to PIP3
7. Activation of Akt
8. Glut4 receptors transported to cell membrane
Answer:
Enzyme inhibitors may alter the elimination rate of a particular drug or inhibit detoxication pathways
Explanation:
An enzyme inhibitor is a chemical compound capable of binding to an enzyme in order to reduce its activity. An enzyme inhibitor may bind to an active site and thus decreases the rate of reaction (direct inhibition). Enzyme inhibitors may act 1-to Inhibit detoxification enzymes and 2-reduce the elimination rate of enzyme drugs, thereby having dose-dependent toxicity effects. For example, many medicines are based on the inhibition of Cytochromes P450 (CYPs) proteins, which are a superfamily of enzymes required for drug metabolism.
What on earth, please repeat the question clearer
Answer:
your joints, stretch the joint capsule. Gas is rapidly released, which forms bubbles.
Explanation:
Answer:
The correct answer is E. Arachidonic acid is the raw material for synthesis of eicosanoids.
Explanation:
Arachidonic acid is an essential omega-6 fatty acid formed by a 20-carbon chain with four double bonds. The presence of double bonds causes this molecule to have several sites that can be oxidized, allowing the formation of different lipids with different biological activities.
Arachidonic acid is part of phospholipids in cell membranes, and is the precursor of eicosanoid biosynthesis. Arachidonic acid can be synthesized from linoleic acid, one of the essential fatty acids required by most mammals.
