A glucagon<span>oma </span>may be a terribly<span> rare </span>tumour<span> of the </span>exocrine gland within which there's a rise<span> in </span>unleash<span> of the </span>internal secretion<span> glucagon, </span>inflicting<span> a characteristic </span>efflorescence<span> and raised </span>glucose<span> levels.due to this the </span>glucose<span> level </span>are going to be exaggerated<span>.Diabetes mellitus </span>kind one<span>, </span>additionally referred to as kind one polygenic disorder<span>, </span>may be a sort of diabetes within which<span> not enough </span>insulin<span> is </span>made<span>.[4] This </span>ends up in<span> high </span>glucose<span> levels </span>within the<span> body.where as in glucagonoma high levels of chromogranin A, </span>that may be a macromolecule typically<span> found in </span>tumor<span> tumors, and anemia, </span>that may be a<span> condition </span>within which you've got a low<span> level of red blood cells.</span>
Answer:
Ciliary muscle
Explanation:
Muscle that changes the thickness of the eye lens through contractions and relaxations
Answer:
5) air temperature - corn sprouting time
6) sacks of fertilizer sold - algae growth
7) cars - pollution levels (sorry couldn't read the full problem, but you get it)
8) distance of flash light - size of illuminated area
9) number of answered questions - score of the final
10) minutes of exposure - birth defect rate
11) amount of vitamin A - weight fluctuation
12) fox population - rabbit population
Explanation:
So as the words speak for themselves, dependent variables are variables that aren't changed by outside factors unlike dependent variables which change based on an outside change. Knowing this, if you reread the problems you will be able to understand it a little bit more.
Answer:
BRCA1 and BRCA2 genes
Explanation:
The BRCA1 and BRCA2 genes are tumor suppressor genes that encode breast cancer susceptibility proteins involved in repairing double-strand DNA breaks (DSBs) by the mechanism of homologous recombination. This mechanism uses the sister chromatid of the homologous chromosome as template for repairing DSBs. It has been shown that mutations in BRCA1 and BRCA2 genes alter the molecular mechanism of homologous recombination DNA repair, thereby mutations are not repaired appropriately and persist in the DNA of tumor cells.
