Hello! I figured your question was lacking its options so I went online to find them and here they are:
A. Activated platelets release chemicals that inhibit blood clot formation.
B. Activated platelets release signaling molecules that inhibit cell division in damaged tissue.
C. Activated platelets constrict the blood vessels, stopping blood flow.
D. Activated platelets release chemicals that activate more platelets.
Answer:
The correct answer is: D) Activated platelets release chemicals that activate more platelets.
Explanation:
<u>Positive feedback</u><u> is a mechanism in which a stimulus triggers an intensification of this same stimulus.</u>
When a tissue is damaged, the platelets participate in a process called platelet adhesion that consists of contacting and sticking to the collagen fibers of the connective tissue under the damaged endothelial cells. This platelet adhesion <u>activates</u> the platelets and makes them grow projections to contact and interact with one another, while releasing substances to activate other nearby platelets. The accumulation and the attachment of large numbers of platelets ultimately form a <u>platelet plug</u>.
The order is calcification of matrix >> cells differentiate into osteoblasts >> formation of the primary ossification >> osteoclasts break down the spongy bone >> formation of the secondary ossification (5,3,1,2,4). It is a fundamental process.
<h3>What are osteoblasts?</h3>
Osteoblasts are cells of the bones, which act to generate bone matrix and modulate the process of mineralization of the skeleton.
Endochondral ossification refers to the mechanism through which the cartilaginous bones generate longitudinal growth.
This mechanism (endochondral ossification) is fundamental during fetal/embryo development.
Learn more about endochondral ossification here:
brainly.com/question/5325975
Minerals move downward through soils in a process called leaching.
God bless!
Your answer would be photosynthesis because photosynthesis synthesis carbon based energy molecules from chloroplast inside a cell, with the outer membrance.
hope i helped my fellow brainily user :)
Answer:
C
Explanation:
I think its C . A transcriptional repressor usually represses the transcription pathway when its active. According to the question, the repressor is not usually active until an effector molecule binds to it making it active and blocking the transcription pathway. So if the region where the effector binds on the repressor is mutated i.e. it turns nonfunctional that means the effector cannot bind to repressor which means repressor cannot become active to block transcription which in turn increases the transcription of gene A because repressor cannot repress it since it is inactive due to its inability to bind to the effector.
ALOT of words please lmk if it makes sense
