Answer: Antibiotics targets the synthesis of protein, nucleic acid, folate and cell wall.
1. Synthesis of protein; antibiotics binds to either 30s or 50s ribosomal subunits blocking the polypeptide from the exiting the tunnel thus inhibiting a full completion of protein expression or production.
2. Nucleic acid synthesis; Antibiotics also act by inhibiting genetic expression, DNA transcription and replication where DNA makes exact copies of itself, as well as RNA molecules preventing bacterial growth.
3. Cell wall synthesis; Inhibition of cell wall synthesis in microorganisms will prevent it from replication and growth.
4. Folate synthesis; Folic acid also known as vitamin B9 helps in DNA replication and cell division. Folate antagonists such as aminopterin kills bacteria by preventing folic acid production required for DNA replication.
Any condition in an experiment that can be changed is a control variable!!
You control it!!
Answer:
The Miller-Urey experiments essential to the theory of evolution because they showed how organic molecules could be made from Earth's early atmosphere.
Explanation:
Answer:
A. His mitochondria lack the transport protein that moves pyruvate across the outer mitochondrial membrane.
Explanation:
Pyruvate is from the breakdown of carbohydrates such as glucose through glycolysis. Glucose enters the cytosol through specific transporters (the GLUT family) and is processed by one of several pathways depending on cellular requirements. Glycolysis occurs in the cytosol and produces a limited amount of ATP, but the end product is two 3-carbon molecules of pyruvate, which maybe diverted again into many pathways depending on the requirements of the cell. In aerobic conditions, pyruvate is primarily transported into the mitochondrial matrix and converted to acetyl-coenzyme A (acetyl-CoA) and carbon dioxide by the pyruvate dehydrogenase complex (PDC).
Initially it was proposed that pyruvate was able to cross the membrane in its undissociated (acid) form but evaluation of its biochemical properties show that it is largely in its ionic form within the cell and should therefore require a transporter.
Transport of pyruvate across the outer mitochondrial membrane appears to be easily accomplished via large non-selective channels such as voltage-dependent anion channels/porin, which enable passive diffusion. Indeed, deficiencies in these channels have been suggested to block pyruvate metabolism
